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Results: 1-9 |
Results: 9
Re-expression of estrogen receptor alpha in estrogen receptor alpha-negative MCF-7 cells restores both estrogen and insulin-like growth factor-mediated signaling and growth
Authors: Oesterreich, S Zhang, P Guler, RL Sun, XH Curran, EM Welshons, WV Osborne, CK Lee, AV
Citation: S. Oesterreich et al., Re-expression of estrogen receptor alpha in estrogen receptor alpha-negative MCF-7 cells restores both estrogen and insulin-like growth factor-mediated signaling and growth, CANCER RES, 61(15), 2001, pp. 5771-5777
High rates of loss of heterozygosity on chromosome 19p13 in human breast cancer
Authors: Oesterreich, S Allredl, DC Mohsin, SK Zhang, Q Wong, H Lee, AV Osborne, CK O'Connell, P
Citation: S. Oesterreich et al., High rates of loss of heterozygosity on chromosome 19p13 in human breast cancer, BR J CANC, 84(4), 2001, pp. 493-498
HET/SAF-B overexpression causes growth arrest and multinuclearity and is associated with aneuploidy in human breast cancer
Authors: Townson, SM Sullivan, T Zhang, QP Clark, GM Osborne, CK Lee, AV Oesterreich, S
Citation: Sm. Townson et al., HET/SAF-B overexpression causes growth arrest and multinuclearity and is associated with aneuploidy in human breast cancer, CLIN CANC R, 6(9), 2000, pp. 3788-3796
Inhibition of oestrogen receptor activity by the co-repressor HET/SAF-B isrelieved by blockade of histone deacetylase activity
Authors: Oesterreich, S Zhang, QP Lee, AV
Citation: S. Oesterreich et al., Inhibition of oestrogen receptor activity by the co-repressor HET/SAF-B isrelieved by blockade of histone deacetylase activity, EUR J CANC, 36, 2000, pp. S43-S44
Tamoxifen-bound estrogen receptor (ER) strongly interacts with the nuclearmatrix protein HET/SAF-B, a novel inhibitor of ER-mediated transactivation
Authors: Oesterreich, S Zhang, QP Hopp, T Fuqua, SAW Michaelis, M Zhao, HH Davie, JR Osborne, CK Lee, AV
Citation: S. Oesterreich et al., Tamoxifen-bound estrogen receptor (ER) strongly interacts with the nuclearmatrix protein HET/SAF-B, a novel inhibitor of ER-mediated transactivation, MOL ENDOCR, 14(3), 2000, pp. 369-381
Insulin-like growth factor I-induced degradation of insulin receptor substrate 1 is mediated by the 26S proteasome and blocked by phosphatidylinositol 3 '-kinase inhibition
Authors: Lee, AV Gooch, JL Oesterreich, S Guler, RL Yee, D
Citation: Av. Lee et al., Insulin-like growth factor I-induced degradation of insulin receptor substrate 1 is mediated by the 26S proteasome and blocked by phosphatidylinositol 3 '-kinase inhibition, MOL CELL B, 20(5), 2000, pp. 1489-1496
Tumor suppressor genes in breast cancer
Authors: Oesterreich, S Fuqua, SAW
Citation: S. Oesterreich et Saw. Fuqua, Tumor suppressor genes in breast cancer, ENDOCR-R CA, 6(3), 1999, pp. 405-419
Low cell motility induced by hsp27 overexpression decreases osteolytic bone metastases of human breast cancer cells in vivo
Authors: Lemieux, P Harvey, J Guise, T Dallas, M Oesterreich, S Yin, JJ Selander, K Fuqua, S
Citation: P. Lemieux et al., Low cell motility induced by hsp27 overexpression decreases osteolytic bone metastases of human breast cancer cells in vivo, J BONE MIN, 14(9), 1999, pp. 1570-1575
Hsp27 overexpression inhibits doxorubicin-induced apoptosis in human breast cancer cells
Authors: Hansen, RK Parra, I Lemieux, P Oesterreich, S Hilsenbeck, SG Fuqua, SAW
Citation: Rk. Hansen et al., Hsp27 overexpression inhibits doxorubicin-induced apoptosis in human breast cancer cells, BREAST CANC, 56(2), 1999, pp. 187-196
Risultati: 1-9 |