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Citation: Yp. Zhou et al., BCL-X-L OVEREXPRESSION PREVENTS APOPTOSIS IN PANCREATIC BETA-CELLS, BUT INHIBITS INSULIN-SECRETION AND INDUCES DIABETES, Diabetes, 47, 1998, pp. 1033-1033
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Citation: Dg. Maggs et al., METABOLIC EFFECTS OF TROGLITAZONE MONOTHERAPY IN TYPE-2 DIABETES-MELLITUS - A RANDOMIZED, DOUBLE-BLIND, PLACEBO-CONTROLLED TRIAL, Annals of internal medicine, 128(3), 1998, pp. 176-185
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Citation: Da. Ehrmann et al., TROGLITAZONE IMPROVES DEFECTS IN INSULIN ACTION, INSULIN-SECRETION, OVARIAN STEROIDOGENESIS, AND FIBRINOLYSIS IN WOMEN WITH POLYCYSTIC-OVARY-SYNDROME, The Journal of clinical endocrinology and metabolism, 82(7), 1997, pp. 2108-2116
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Citation: Bn. Cockburn et al., CHANGES IN PANCREATIC-ISLET GLUCOKINASE AND HEXOKINASE ACTIVITIES WITH INCREASING AGE, OBESITY, AND THE ONSET OF DIABETES, Diabetes, 46(9), 1997, pp. 1434-1439
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Citation: Wh. Herman et al., DIMINISHED INSULIN AND GLUCAGON SECRETORY RESPONSES TO ARGININE IN NONDIABETIC SUBJECTS WITH A MUTATION IN THE HEPATOCYTE NUCLEAR FACTOR 4-ALPHA MODY1 GENE/, Diabetes, 46(11), 1997, pp. 1749-1754
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Citation: Mk. Cavaghan et al., TREATMENT WITH THE ORAL ANTIDIABETIC AGENT TROGLITAZONE IMPROVES BETA-CELL RESPONSES TO GLUCOSE IN SUBJECTS WITH IGT, Diabetes, 46, 1997, pp. 834-834