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Results:
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Results: 20
Authors:
SENGPIEL B
PREIS E
KRIEGLSTEIN J
PREHN JHM
Citation: B. Sengpiel et al., NMDA-INDUCED SUPEROXIDE PRODUCTION AND NEUROTOXICITY IN CULTURED RAT HIPPOCAMPAL-NEURONS - ROLE OF MITOCHONDRIA, European journal of neuroscience, 10(5), 1998, pp. 1903-1910
Authors:
KROHN AJ
PREIS E
PREHN JHM
Citation: Aj. Krohn et al., STAUROSPORINE-INDUCED APOPTOSIS OF CULTURED RAT HIPPOCAMPAL-NEURONS INVOLVES CASPASE-1-LIKE PROTEASES AS UPSTREAM INITIATORS AND INCREASED PRODUCTION OF SUPEROXIDE AS A MAIN DOWNSTREAM EFFECTOR, The Journal of neuroscience, 18(20), 1998, pp. 8186-8197
Authors:
PREHN JHM
Citation: Jhm. Prehn, MITOCHONDRIAL TRANSMEMBRANE POTENTIAL AND FREE-RADICAL PRODUCTION IN EXCITOTOXIC NEURODEGENERATION, Naunyn-Schmiedeberg's archives of pharmacology, 357(3), 1998, pp. 316-322
Authors:
JORDAN J
GALINDO MF
PREHN JHM
WEICHSELBAUM RR
BECKETT M
GHADGE GD
ROOS RP
LEIDEN JM
MILLER RJ
Citation: J. Jordan et al., P53 EXPRESSION INDUCES APOPTOSIS IN HIPPOCAMPAL PYRAMIDAL NEURON CULTURES, The Journal of neuroscience, 17(4), 1997, pp. 1397-1405
Authors:
PREHN JHM
Citation: Jhm. Prehn, FREE-RADICAL PRODUCTION IS A KEY EVENT IN PROGRAMMED CELL-DEATH OF CULTURED RAT HIPPOCAMPAL-NEURONS, Naunyn-Schmiedeberg's archives of pharmacology, 356(4), 1997, pp. 51-51
Authors:
PREHN JHM
JORDAN J
GHADGE GD
PREIS E
GALINDO MF
ROOS RP
KRIEGLSTEIN J
MILLER RJ
Citation: Jhm. Prehn et al., CA2-INDUCED NEURONAL APOPTOSIS( AND REACTIVE OXYGEN SPECIES IN STAUROSPORINE), Journal of neurochemistry, 68(4), 1997, pp. 1679-1685
Authors:
WIND T
PREHN JHM
PERUCHE B
KRIEGLSTEIN J
Citation: T. Wind et al., ACTIVATION OF ATP-SENSITIVE POTASSIUM CHANNELS DECREASES NEURONAL INJURY CAUSED BY CHEMICAL HYPOXIA, Brain research, 751(2), 1997, pp. 295-299
Authors:
HENRICHNOACK P
PREHN JHM
KRIEGLSTEIN J
Citation: P. Henrichnoack et al., TGF-BETA-1 PROTECTS HIPPOCAMPAL-NEURONS AGAINST DEGENERATION CAUSED BY TRANSIENT GLOBAL-ISCHEMIA - DOSE-RESPONSE RELATIONSHIP AND POTENTIALNEUROPROTECTIVE MECHANISMS, Stroke, 27(9), 1996, pp. 1609-1614
Authors:
PREHN JHM
MILLER RJ
Citation: Jhm. Prehn et Rj. Miller, OPPOSITE EFFECTS OF TGF-BETA-1 ON RAPIDLY-TRIGGERED AND SLOWLY-TRIGGERED EXCITOTOXIC INJURY, Neuropharmacology, 35(3), 1996, pp. 249-256
Authors:
PREHN JHM
BINDOKAS VP
JORDAN J
GALINDO MF
GHADGE GD
ROOS RP
BOISE LH
THOMPSON CB
KRAJEWSKI S
REED JC
MILLER RJ
Citation: Jhm. Prehn et al., PROTECTIVE EFFECT OF TRANSFORMING GROWTH-FACTOR-BETA-1 ON BETA-AMYLOID NEUROTOXICITY IN RAT HIPPOCAMPAL-NEURONS, Molecular pharmacology, 49(2), 1996, pp. 319-328
Authors:
PREHN JHM
Citation: Jhm. Prehn, MARKED DIVERSITY IN THE ACTION OF GROWTH-FACTORS ON N-METHYL-D-ASPARTATE-INDUCED NEURONAL DEGENERATION, European journal of pharmacology, 306(1-3), 1996, pp. 81-88
Authors:
PREHN JHM
LIPPERT K
KRIEGLSTEIN J
Citation: Jhm. Prehn et al., ARE NMDA OR AMPA KAINATE RECEPTOR ANTAGONISTS MORE EFFICACIOUS IN THEDELAYED TREATMENT OF EXCITOTOXIC NEURONAL INJURY, European journal of pharmacology. Environmental toxicology and pharmacology section, 292(2), 1995, pp. 179-189
Authors:
JORDAN J
GHADGE GD
PREHN JHM
TOTH PT
ROOS RP
MILLER RJ
Citation: J. Jordan et al., EXPRESSION OF HUMAN COPPER ZINC-SUPEROXIDE DISMUTASE INHIBITS THE DEATH OF RAT SYMPATHETIC NEURONS CAUSED BY WITHDRAWAL OF NERVE GROWTH-FACTOR/, Molecular pharmacology, 47(6), 1995, pp. 1095-1100
Authors:
PERUCHE B
BACKHAUSS C
PREHN JHM
KRIEGLSTEIN J
Citation: B. Peruche et al., PROTECTIVE EFFECTS OF 5-HT1A RECEPTOR AGONISTS AGAINST NEURONAL DAMAGE DEMONSTRATED IN-VIVO AND IN-VITRO, Journal of neural transmission. Parkinson's disease and dementia section, 8(1-2), 1994, pp. 73-83
Authors:
PREHN JHM
KRIEGLSTEIN J
Citation: Jhm. Prehn et J. Krieglstein, OPPOSING EFFECTS OF TRANSFORMING GROWTH-FACTOR-BETA-1 ON GLUTAMATE NEUROTOXICITY, Neuroscience, 60(1), 1994, pp. 7-10
Authors:
HENRICHNOACK P
PREHN JHM
KRIEGLSTEIN J
Citation: P. Henrichnoack et al., NEUROPROTECTIVE EFFECTS OF TGF-BETA-1, Journal of neural transmission. Supplementum, 1994, pp. 33-45
Authors:
PREHN JHM
BINDOKAS VP
MARCUCCILLI CJ
KRAJEWSKI S
REED JC
MILLER RJ
Citation: Jhm. Prehn et al., REGULATION OF NEURONAL BCL2 PROTEIN EXPRESSION AND CALCIUM HOMEOSTASIS BY TRANSFORMING GROWTH-FACTOR TYPE-BETA CONFERS WIDE-RANGING PROTECTION ON RAT HIPPOCAMPAL-NEURONS, Proceedings of the National Academy of Sciences of the United Statesof America, 91(26), 1994, pp. 12599-12603
Authors:
PREHN JHM
BACKHAUSS C
KRIEGLSTEIN J
Citation: Jhm. Prehn et al., TRANSFORMING GROWTH-FACTOR-BETA-1 PREVENTS GLUTAMATE NEUROTOXICITY INRAT NEOCORTICAL CULTURES AND PROTECTS MOUSE NEOCORTEX FROM ISCHEMIC-INJURY INVIVO, Journal of cerebral blood flow and metabolism, 13(3), 1993, pp. 521-525
Authors:
PREHN JHM
PERUCHE B
UNSICKER K
KRIEGLSTEIN J
Citation: Jhm. Prehn et al., ISOFORM-SPECIFIC EFFECTS OF TRANSFORMING GROWTH FACTORS-BETA ON DEGENERATION OF PRIMARY NEURONAL CULTURES INDUCED BY CYTOTOXIC HYPOXIA OR GLUTAMATE, Journal of neurochemistry, 60(5), 1993, pp. 1665-1672
Authors:
PREHN JHM
WELSCH M
BACKHAUSS C
NUGLISCH J
AUSMEIER F
KARKOUTLY C
KRIEGLSTEIN J
Citation: Jhm. Prehn et al., EFFECTS OF SEROTONERGIC DRUGS IN EXPERIMENTAL BRAIN ISCHEMIA - EVIDENCE FOR A PROTECTIVE ROLE OF SEROTONIN IN CEREBRAL-ISCHEMIA, Brain research, 630(1-2), 1993, pp. 10-20
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