Citation: Rs. Fischer et al., RAC1 AND EXTRACELLULARLY REGULATED KINASE ACTIVATION ARE SUFFICIENT FOR E1A-DEPENDENT COOPERATIVE TRANSFORMATION OF PRIMARY EPITHELIAL-CELLS, BUT PROGRESSION CAN ONLY BE MODULATED BY E1A OR RAC1, Cell growth & differentiation, 9(3), 1998, pp. 209-221
Citation: Rs. Fischer et Mp. Quinlan, IDENTIFICATION OF A NOVEL MECHANISM OF REGULATION OF THE ADHERENS JUNCTION BY E1A, RAC1, AND CORTICAL ACTIN-FILAMENTS THAT CONTRIBUTES TO TUMOR PROGRESSION, Cell growth & differentiation, 9(11), 1998, pp. 905-918
Citation: Rs. Fischer et Mp. Quinlan, THE C-TERMINUS OF E1A REGULATES TUMOR PROGRESSION AND EPITHELIAL-CELLDIFFERENTIATION, Virology (New York, N.Y. Print), 249(2), 1998, pp. 427-439
Citation: Rs. Fischer et Mp. Quinlan, EXPRESSION OF THE PRB-BINDING REGIONS OF E1A ENABLES EFFICIENT TRANSFORMATION OF PRIMARY EPITHELIAL-CELLS BY V-SRC, Journal of virology, 72(4), 1998, pp. 2815-2824
Citation: S. Gopalakrishnan et al., IMMORTALIZATION OF PRIMARY EPITHELIAL-CELLS BY E1A 12S REQUIRES LATE,2ND EXON-ENCODED FUNCTIONS IN ADDITION TO COMPLEX-FORMATION WITH PRB AND P300, Cell growth & differentiation, 8(5), 1997, pp. 541-551
Citation: A. Malladi et Mp. Quinlan, MUTATIONS IN CR-1 OF E1A 12S YIELD DOMINANT-NEGATIVE SUPPRESSORS OF IMMORTALIZATION AND THE LYTIC CYCLE, Virology, 233(1), 1997, pp. 51-62
Citation: Lm. Digiovanni et al., CHOROID-PLEXUS CYSTS - INFANT AND EARLY-CHILDHOOD DEVELOPMENTAL OUTCOME, Obstetrics and gynecology, 90(2), 1997, pp. 191-194
Citation: Mp. Quinlan et Rs. Fischer, LOSS OF DIFFERENTIATION CAUSES ENHANCED TUMOR PROGRESSION OF EPITHELIAL-CELLS IN THE PRESENCE OF ACTIVATED SMALL GTPASES AND HYPERTRANSFORMING E1A MUTANTS, Molecular biology of the cell, 7, 1996, pp. 2856-2856
Citation: Rs. Fischer et Mp. Quinlan, RAC CDC42 INVOLVEMENT IN RAS HYPERTRANSFORMATION OF PRIMARY EPITHELIAL-CELLS WITH E1A/, Molecular biology of the cell, 7, 1996, pp. 2988-2988
Citation: S. Gopalakrishnan et al., INDUCTION OF A COMPLEX BETWEEN RASGAP AND A NOVEL 110 KD PROTEIN IS REQUIRED FOR IMMORTALIZATION OF PRIMARY EPITHELIAL-CELLS BY THE E1A 12SONCOPROTEIN OF ADENOVIRUS, Oncogene, 13(12), 1996, pp. 2659-2669
Citation: Ck. Krantz et al., E1A 2ND EXON REQUIREMENTS FOR INDUCTION OF TARGET-CELL SUSCEPTIBILITYTO LYSIS BY NATURAL-KILLER-CELLS - IMPLICATIONS FOR THE MECHANISM OF ACTION, Virology, 217(1), 1996, pp. 23-32
Citation: S. Gopalakrishnan et Mp. Quinlan, INDUCTION OF GAP COMPLEXES DURING IMMORTALIZATION OF PRIMARY EPITHELIAL-CELLS BY ADENOVIRUS E1A 12S PROTEIN, Molecular biology of the cell, 6, 1995, pp. 2013-2013
Citation: S. Gopalakrishnan et Mp. Quinlan, MODULATION OF E-CADHERIN LOCALIZATION IN CELLS EXPRESSING WILD-TYPE E1A 12S OR HYPERTRANSFORMING MUTANTS, Cell growth & differentiation, 6(8), 1995, pp. 985-998
Citation: Jl. Douglas et Mp. Quinlan, EFFICIENT NUCLEAR-LOCALIZATION AND IMMORTALIZING ABILITY, 2 FUNCTIONSDEPENDENT ON THE ADENOVIRUS-TYPE-5 (AD5) E1A 2ND EXON, ARE NECESSARY FOR COTRANSFORMATION WITH AD5 E1B BUT NOT WITH T24RAS, Journal of virology, 69(12), 1995, pp. 8061-8065
Citation: Jl. Douglas et Mp. Quinlan, EFFICIENT NUCLEAR-LOCALIZATION OF THE AD5 E1A 12S PROTEIN IS NECESSARY FOR IMMORTALIZATION BUT NOT COTRANSFORMATION OF PRIMARY EPITHELIAL-CELLS, Cell growth & differentiation, 5(5), 1994, pp. 475-483
Citation: Mp. Quinlan, ENHANCED PROLIFERATION, GROWTH-FACTOR INDUCTION AND IMMORTALIZATION BY ADENOVIRUS E1A 12S IN THE ABSENCE OF E1B, Oncogene, 9(9), 1994, pp. 2639-2647
Citation: Mp. Quinlan, E1A 12S IN THE ABSENCE OF E1B OR OTHER COOPERATING ONCOGENES ENABLES CELLS TO OVERCOME APOPTOSIS, Oncogene, 8(12), 1993, pp. 3289-3296