Authors:
RAMJEESINGH M
HUAN LJ
WILSCHANSKI M
DURIE P
LI CH
GYOMOREY K
WANG YC
KENT G
TANSWELL KA
CUTZ E
ACKERLEY C
BEAR CE
Citation: M. Ramjeesingh et al., ASSESSMENT OF THE EFFICACY OF IN-VIVO CFTR PROTEIN REPLACEMENT THERAPY IN CF MICE, Human gene therapy, 9(4), 1998, pp. 521-528
Authors:
GYOMOREY K
YEGER H
ROZMAHEL R
GALLEY KA
GARAMI E
RAMJEESINGH M
ROMMENS JM
TSUI LC
BEAR CE
Citation: K. Gyomorey et al., CONTRIBUTION OF CLC-2 TO INTESTINAL CL- SECRETION IN THE GASTROINTESTINAL-TRACT OF CYSTIC-FIBROSIS MICE, The Journal of general physiology, 112(1), 1998, pp. 67-67
Authors:
RAMJEESINGH M
LI CH
GARAMI E
HUAN LJ
HEWRYK M
WANG YC
GALLEY K
BEAR CE
Citation: M. Ramjeesingh et al., A NOVEL PROCEDURE FOR THE EFFICIENT PURIFICATION OF THE CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR (CFTR), Biochemical journal, 327, 1997, pp. 17-21
Authors:
BEAR CE
LI CH
GALLEY K
WANG YC
GARAMI E
RAMJEESINGH M
Citation: Ce. Bear et al., COUPLING OF ATP HYDROLYSIS WITH CHANNEL GATING BY PURIFIED, RECONSTITUTED CFTR, Journal of bioenergetics and biomembranes, 29(5), 1997, pp. 465-473
Authors:
LI CH
RAMJEESINGH M
WANG W
GARAMI E
HEWRYK M
LEE D
ROMMENS JM
GALLEY K
BEAR CE
Citation: Ch. Li et al., ATPASE ACTIVITY OF THE CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR, The Journal of biological chemistry, 271(45), 1996, pp. 28463-28468
Citation: Ch. Li et al., PURIFIED CYSTIC-FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR (CFTR) DOES NOT FUNCTION AS AN ATP CHANNEL, The Journal of biological chemistry, 271(20), 1996, pp. 11623-11626
Authors:
NEWSTED WJ
RAMJEESINGH M
ZYWULKO M
ROTHSTEIN SJ
SHAMI EY
Citation: Wj. Newsted et al., ENGINEERING RESISTANCE TO TRYPSIN INACTIVATION INTO L-ASPARAGINASE THROUGH THE PRODUCTION OF A CHIMERIC PROTEIN BETWEEN THE ENZYME AND A PROTECTIVE SINGLE-CHAIN ANTIBODY, Enzyme and microbial technology, 17(8), 1995, pp. 757-764
Authors:
SHAMI EY
NEWSTED WJ
RAMJEESINGH M
ZYWULKO M
ROTHSTEIN SJ
Citation: Ey. Shami et al., CHIMERIC ESCHERICHIA-COLI L-ASPARAGINASE II RESISTANT TO INACTIVATIONBY TRYPSIN WAS CONSTRUCTED BY GENETICALLY FUSING A SINGLE-CHAIN ANTIBODY (DERIVED FROM A PROTECTIVE MAB) TO THE ENZYME MONOMER, Journal of cellular biochemistry, 1994, pp. 217-217