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Results: 1-10 |
Results: 10
MALONONITRILAMIDES (MNAS) IN COMBINATION WITH CYCLOSPORINE SYNERGISTICALLY PREVENT ACUTE AND TREAT ONGOING SKIN ALLOGRAFT-REJECTION
Authors: SCHORLEMMER HU RUUTH E KURRLE R
Citation: Hu. Schorlemmer et al., MALONONITRILAMIDES (MNAS) IN COMBINATION WITH CYCLOSPORINE SYNERGISTICALLY PREVENT ACUTE AND TREAT ONGOING SKIN ALLOGRAFT-REJECTION, International journal of immunotherapy, 13(1-2), 1997, pp. 9-16
THE ALLOREACTIVITY IN THE POPLITEAL LYMPH-NODE (PLN) ASSAY IS REGULATED BY MALONONITRILAMIDES (MNAS)
Authors: SCHORLEMMER HU RUUTH E KURRLE R
Citation: Hu. Schorlemmer et al., THE ALLOREACTIVITY IN THE POPLITEAL LYMPH-NODE (PLN) ASSAY IS REGULATED BY MALONONITRILAMIDES (MNAS), International journal on tissue reactions, 19(3-4), 1997, pp. 157-161
MALONONITRILAMIDES INHIBIT T-CELL AND B-CELL RESPONSIVENESS IN DIFFERENT SPECIES
Authors: KURRLE R RUUTH E BARTLETT R LAUFFER L SCHORLEMMER HU
Citation: R. Kurrle et al., MALONONITRILAMIDES INHIBIT T-CELL AND B-CELL RESPONSIVENESS IN DIFFERENT SPECIES, Transplantation proceedings, 29(1-2), 1997, pp. 1302-1303
POTENCIES OF LEFLUNOMIDE AND HR325 AS INHIBITORS OF PROSTAGLANDIN-ENDOPEROXIDE-H SYNTHASE-1 AND SYNTHASE-2 - COMPARISON WITH NONSTEROIDAL ANTIINFLAMMATORY DRUGS
Authors: CURNOCK AP ROBSON PA YEA CM MOSS D GADHER S THOMSON TA WESTWOOD R RUUTH E WILLIAMSON RA
Citation: Ap. Curnock et al., POTENCIES OF LEFLUNOMIDE AND HR325 AS INHIBITORS OF PROSTAGLANDIN-ENDOPEROXIDE-H SYNTHASE-1 AND SYNTHASE-2 - COMPARISON WITH NONSTEROIDAL ANTIINFLAMMATORY DRUGS, The Journal of pharmacology and experimental therapeutics, 282(1), 1997, pp. 339-347
TREATMENT WITH NEUTRALIZING ANTIBODIES SPECIFIC FOR IL-1-BETA PREVENTS CYCLOPHOSPHAMIDE-INDUCED DIABETES IN NONOBESE DIABETIC MICE
Authors: CAILLEAU C DIUHERCEND A RUUTH E WESTWOOD R CARNAUD C
Citation: C. Cailleau et al., TREATMENT WITH NEUTRALIZING ANTIBODIES SPECIFIC FOR IL-1-BETA PREVENTS CYCLOPHOSPHAMIDE-INDUCED DIABETES IN NONOBESE DIABETIC MICE, Diabetes, 46(6), 1997, pp. 937-940
ACUTE SKIN-GRAFT REJECTION CAN BE PREVENTED AND TREATED IN RAT MODELSBY MALONONITRILAMIDES
Authors: SCHORLEMMER HU SCHWAB W RUUTH E KURRLE R
Citation: Hu. Schorlemmer et al., ACUTE SKIN-GRAFT REJECTION CAN BE PREVENTED AND TREATED IN RAT MODELSBY MALONONITRILAMIDES, Transplantation proceedings, 28(6), 1996, pp. 3048-3050
MALONONITRILAMIDES INHIBIT T-CELL AND B-CELL RESPONSIVENESS
Authors: KURRLE R BARTLETT R RUUTH E LAUFFER L SCHORLEMMER HU
Citation: R. Kurrle et al., MALONONITRILAMIDES INHIBIT T-CELL AND B-CELL RESPONSIVENESS, Transplantation proceedings, 28(6), 1996, pp. 3053-3056
DIHYDROOROTATE DEHYDROGENASE IS A TARGET FOR THE BIOLOGICAL EFFECTS OF LEFLUNOMIDE
Authors: WILLIAMSON RA YEA CM ROBSON PA CURNOCK AP GADHER S HAMBLETON AB WOODWARD K BRUNEAU JM HAMBLETON P SPINELLAJAEGLE S MORAND P COURTIN O SAUTES C WESTWOOD R HERCEND T KUO EA RUUTH E
Citation: Ra. Williamson et al., DIHYDROOROTATE DEHYDROGENASE IS A TARGET FOR THE BIOLOGICAL EFFECTS OF LEFLUNOMIDE, Transplantation proceedings, 28(6), 1996, pp. 3088-3091
DIHYDROOROTATE DEHYDROGENASE IS A HIGH-AFFINITY BINDING-PROTEIN FOR A77-1726 AND MEDIATOR OF A RANGE OF BIOLOGICAL EFFECTS OF THE IMMUNOMODULATORY COMPOUND
Authors: WILLIAMSON RA YEA CM ROBSON PA CURNOCK AP GADHER S HAMBLETON AB WOODWARD K BRUNEAU JM HAMBLETON P MOSS D THOMSON TA SPINELLAJAEGLE S MORAND P COURTIN O SAUTES C WESTWOOD R HERCEND T KUO EA RUUTH E
Citation: Ra. Williamson et al., DIHYDROOROTATE DEHYDROGENASE IS A HIGH-AFFINITY BINDING-PROTEIN FOR A77-1726 AND MEDIATOR OF A RANGE OF BIOLOGICAL EFFECTS OF THE IMMUNOMODULATORY COMPOUND, The Journal of biological chemistry, 270(38), 1995, pp. 22467-22472
MYCOPLASMA-ARGININI TUH-14 MEMBRANE LIPOPROTEINS INDUCE PRODUCTION OFINTERLEUKIN-1, INTERLEUKIN-6, AND TUMOR-NECROSIS-FACTOR-ALPHA BY HUMAN MONOCYTES
Authors: HERBELIN A RUUTH E DELORME D MICHELHERBELIN C PRAZ F
Citation: A. Herbelin et al., MYCOPLASMA-ARGININI TUH-14 MEMBRANE LIPOPROTEINS INDUCE PRODUCTION OFINTERLEUKIN-1, INTERLEUKIN-6, AND TUMOR-NECROSIS-FACTOR-ALPHA BY HUMAN MONOCYTES, Infection and immunity, 62(10), 1994, pp. 4690-4694
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