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Citation: Bi. Razzouk et al., Impact of treatment on the outcome of acute myeloid leukemia with inversion 16: a single institution's experience, LEUKEMIA, 15(9), 2001, pp. 1326-1330
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Citation: Rm. Slater et al., t(7;12)(q36;p13) and t(7;12)(q32;p13) - translocations involving ETV6 in children 18 months of age or younger with myeloid disorders, LEUKEMIA, 15(6), 2001, pp. 915-920
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Citation: Ra. Krance et al., Experience with 2-chlorodeoxyadenosine in previously untreated children with newly diagnosed acute myeloid leukemia and myelodysplastic diseases, J CL ONCOL, 19(11), 2001, pp. 2804-2811
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Citation: Jg. Blanco et al., Molecular emergence of acute myeloid leukemia during treatment for acute lymphoblastic leukemia, P NAS US, 98(18), 2001, pp. 10338-10343
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Citation: Uh. Athale et al., Biology and outcome of childhood acute megakaryoblastic leukemia: a singleinstitution's experience, BLOOD, 97(12), 2001, pp. 3727-3732
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Citation: S. Mathew et al., Concurrent translocations of MLL and CBFA2 (AMLI) genes with new partner breakpoints in a child with secondary myelodysplastic syndrome after treatment of acute lymphoblastic leukemia, GENE CHROM, 28(2), 2000, pp. 227-232
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Citation: S. Mathew et al., Trisomy of the long arm of chromosome 1 resulting in a dicentric derivative (6)t(1;6) chromosome in a child with myelodysplastic syndrome following treatment for a primitive neuroectodermal tumor, LEUK LYMPH, 37(1-2), 2000, pp. 213
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Citation: M. Chang et al., Prognostic factors in children and adolescents with acute myeloid leukemia(excluding children with Down syndrome and acute promyelocytic leukemia): univariate and recursive partitioning analysis of patients treated on Pediatric Oncology Group (POC) Study 8821, LEUKEMIA, 14(7), 2000, pp. 1201-1207
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Citation: S. Mathew et al., Double minute chromosomes and c-MYC amplification in a child with secondary myelodysplastic syndrome after treatment for acute lymphoblastic leukemia, LEUKEMIA, 14(7), 2000, pp. 1314-1315
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Citation: Mh. Woo et al., Glutathione S-transferase genotypes in children who develop treatment-related acute myeloid malignancies, LEUKEMIA, 14(2), 2000, pp. 232-237
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Citation: Ch. Pui et al., Long-term results of Total Therapy studies 11, 12 and 13A for childhood acute lymphoblastic leukemia at St Jude Children's Research Hospital, LEUKEMIA, 14(12), 2000, pp. 2286-2294
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Citation: Mk. Arnaout et al., Treatment of childhood acute myelogenous leukemia with an intensive regimen (AML-87) that individualizes etoposide and cytarabine dosages: short- andlong-term effects, LEUKEMIA, 14(10), 2000, pp. 1736-1742
Citation: Sc. Raimondi, Fluorescence in situ hybridization: Molecular probes for diagnosis of pediatric neoplastic diseases, CANCER INV, 18(2), 2000, pp. 135-147
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Citation: Se. Morgan et al., Selection of human leukemic CEM cells for resistance to the DNA topoisomerase II catalytic inhibitor ICRF-187 results in increased levels of topoisomerase II alpha and altered G(2)/M checkpoint and apoptotic responses, MOLEC PHARM, 57(2), 2000, pp. 296-307
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Citation: E. Coustan-smith et al., Clinical importance of minimal residual disease in childhood acute lymphoblastic leukemia, BLOOD, 96(8), 2000, pp. 2591-2596
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Citation: E. Coustan-smith et al., Clinical importance of minimal residual disease in childhood acute lymphoblastic leukemia, BLOOD, 96(8), 2000, pp. 2691-2696
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Citation: Sd. Raynaud et al., Cytogenetic abnormalities associated with the t(12;21): a collaborative study of 169 children with t(12;21)-positive acute lymphoblastic leukemia, LEUKEMIA, 13(9), 1999, pp. 1325-1330
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Citation: S. Mathew et al., Comparison of cytogenetics, Southern blotting, and fluorescence in situ hybridization as methods for detecting MLL gene rearrangements in children with acute leukemia and with 11q23 abnormalities, LEUKEMIA, 13(11), 1999, pp. 1713-1720
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Citation: Je. Rubnitz et al., Childhood acute lymphoblastic leukemia with the MLL-ENL fusion and t(11;19)(q23;p13.3) translocation, J CL ONCOL, 17(1), 1999, pp. 191-196