Authors:
Caruso, JA
Reiners, JJ
Emond, J
Shultz, T
Tainsky, MA
Alaoui-Jamali, M
Batist, G
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Authors:
Reiners, JJ
Mathieu, P
Okafor, C
Putt, DA
Lash, LH
Citation: Jj. Reiners et al., Depletion of cellular glutathione by conditions used for the passaging of adherent cultured cells, TOX LETT, 115(2), 2000, pp. 153-163
Citation: M. Guo et Jj. Reiners, Phorbol ester-induced production of cytostatic factors by normal and oncogenic Ha-ras-transformed human breast cell lines (vol 21, pg 1303, 2000), CARCINOGENE, 21(9), 2000, pp. 1767-1767
Citation: M. Guo et Jj. Reiners, Phorbol ester-induced production of cytostatic factors by normal and oncogenic Ha-ras-transformed human breast cell lines, CARCINOGENE, 21(7), 2000, pp. 1303-1312
Authors:
McCabe, MJ
Singh, KP
Reddy, SA
Chelladurai, B
Pounds, JG
Reiners, JJ
States, JC
Citation: Mj. Mccabe et al., Sensitivity of myelomonocytic leukemia cells to arsenite-induced cell cycle disruption, apoptosis, and enhanced differentiation is dependent on the inter-relationship between arsenic concentration, duration of treatment, andcell cycle phase, J PHARM EXP, 295(2), 2000, pp. 724-733
Citation: M. Guo et al., Suppression of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-mediated aryl hydrocarbon receptor transformation and CYP1A1 induction by the phosphatidylinositol 3-kinase inhibitor 2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one(LY294002), BIOCH PHARM, 60(5), 2000, pp. 635-642
Citation: Mj. Mccabe et al., Lead intoxication impairs the generation of a delayed type hypersensitivity response, TOXICOLOGY, 139(3), 1999, pp. 255-264
Citation: Jj. Reiners et al., Suppression of cell cycle progression by flavonoids: dependence on the aryl hydrocarbon receptor, CARCINOGENE, 20(8), 1999, pp. 1561-1566
Citation: Jj. Reiners et Re. Clift, Aryl hydrocarbon receptor regulation of ceramide-induced apoptosis in murine hepatoma 1c1c7 cells - A function independent of aryl hydrocarbon receptor nuclear translocator, J BIOL CHEM, 274(4), 1999, pp. 2502-2510