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Results: 1-7 |
Results: 7

Authors: Vilin, YY Fujimoto, E Ruben, PC
Citation: Yy. Vilin et al., A novel mechanism associated with idiopathic ventricular fibrillation (IVF) mutations R1232W and T1620M in human cardiac sodium channels, PFLUG ARCH, 442(2), 2001, pp. 204-211

Authors: Vilin, YY Fujimoto, E Ruben, PC
Citation: Yy. Vilin et al., A single residue differentiates between human cardiac and skeletal muscle Na+ channel slow inactivation, BIOPHYS J, 80(5), 2001, pp. 2221-2230

Authors: Groome, JR Fujimoto, E Ruben, PC
Citation: Jr. Groome et al., The delay in recovery from fast inactivation in skeletal muscle sodium channels is deactivation, CELL MOL N, 20(4), 2000, pp. 521-527

Authors: Cha, A Ruben, PC George, AL Fujimoto, E Bezanilla, F
Citation: A. Cha et al., Voltage sensors in domains III and IV, but not I and II, are immobilized by Na+ channel fast inactivation, NEURON, 22(1), 1999, pp. 73-87

Authors: Groome, JR Fujimoto, E George, AL Ruben, PC
Citation: Jr. Groome et al., Differential effects of homologous S4 mutations in human skeletal muscle sodium channels on deactivation gating from open and inactivated states, J PHYSL LON, 516(3), 1999, pp. 687-698

Authors: Wang, DW VanDeCarr, D Ruben, PC George, AL Bennett, PB
Citation: Dw. Wang et al., Functional consequences of a domain 1/S6 segment sodium channel mutation associated with painful congenital myotonia, FEBS LETTER, 448(2-3), 1999, pp. 231-234

Authors: Vilin, YY Makita, N George, AL Ruben, PC
Citation: Yy. Vilin et al., Structural determinants of slow inactivation in human cardiac and skeletalmuscle sodium channels, BIOPHYS J, 77(3), 1999, pp. 1384-1393
Risultati: 1-7 |