Authors:
WINSLOW DL
GARBER S
REID C
SCARNATI H
BAKER D
RAYNER MM
ANTON ED
Citation: Dl. Winslow et al., SELECTION CONDITIONS AFFECT THE EVOLUTION OF SPECIFIC MUTATIONS IN THE REVERSE-TRANSCRIPTASE GENE ASSOCIATED WITH RESISTANCE TO DMP-266, AIDS, 10(11), 1996, pp. 1205-1209
Authors:
WINSLOW DL
GARBER S
REID C
SCARNATI H
KORANT B
EMINI E
ANTON ED
Citation: Dl. Winslow et al., DEVELOPMENT OF HIGH-LEVEL RESISTANCE TO DMP-266 REQUIRES MULTIPLE MUTATIONS IN THE REVERSE-TRANSCRIPTASE GENE, Journal of acquired immune deficiency syndromes and human retrovirology, 10, 1995, pp. 13-13
Authors:
ANTON ED
BACHELER L
GARBER S
REID C
BUCKERY R
SCARNATI H
KORANT B
WINSLOW DL
Citation: Ed. Anton et al., REPLICATIVE ABILITY OF HIV VARIANTS WITH SUBSTITUTIONS IN THE ACTIVE-SITE OF PROTEASE IS DEPENDENT ON COMPENSATORY MUTATIONS OUTSIDE THE ACTIVE-SITE, Journal of acquired immune deficiency syndromes and human retrovirology, 10, 1995, pp. 60-60
Authors:
WINSLOW DL
STACK S
KING R
SCARNATI H
BINCSIK A
OTTO MJ
Citation: Dl. Winslow et al., LIMITED SEQUENCE DIVERSITY OF THE HIV TYPE-1 PROTEASE GENE FROM CLINICAL ISOLATES AND IN-VITRO SUSCEPTIBILITY TO HIV PROTEASE INHIBITORS, AIDS research and human retroviruses, 11(1), 1995, pp. 107-113
Authors:
WINSLOW DL
MAYERS D
SCARNATI H
LANE J
BINCSIK A
OTTO MJ
Citation: Dl. Winslow et al., IN-VITRO SUSCEPTIBILITY OF CLINICAL ISOLATES OF HIV-1 TO XM323, A NON-PEPTIDYL HIV PROTEASE INHIBITOR, AIDS, 8(6), 1994, pp. 753-756
Authors:
WINSLOW DL
ANTON ED
HORLICK RA
ZAGURSKY RJ
TRITCH RJ
SCARNATI H
ACKERMAN K
BACHELER LT
Citation: Dl. Winslow et al., CONSTRUCTION OF INFECTIOUS MOLECULAR CLONES OF HIV-1 CONTAINING DEFINED MUTATIONS IN THE PROTEASE GENE, Biochemical and biophysical research communications, 205(3), 1994, pp. 1651-1657
Authors:
OTTO MJ
REID CD
GARBER S
LAM PYS
SCARNATI H
BACHELER LT
RAYNER MM
WINSLOW DL
Citation: Mj. Otto et al., IN-VITRO ANTI-HUMAN-IMMUNODEFICIENCY-VIRUS (HIV) ACTIVITY OF XM323, ANOVEL HIV PROTEASE INHIBITOR, Antimicrobial agents and chemotherapy, 37(12), 1993, pp. 2606-2611