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Results: 1-25 | 26-50 | 51-75 | 76-79
Results: 26-50/79
PHOSPHOLIPIDS ALTER TAU-CONFORMATION, PHOSPHORYLATION, PROTEOLYSIS AND ASSOCIATION WITH MICROTUBULES - IMPLICATION FOR TAU-FUNCTION UNDER NORMAL AND DEGENERATIVE CONDITIONS
Authors: SHEA TB CHANG M
Citation: Tb. Shea et M. Chang, PHOSPHOLIPIDS ALTER TAU-CONFORMATION, PHOSPHORYLATION, PROTEOLYSIS AND ASSOCIATION WITH MICROTUBULES - IMPLICATION FOR TAU-FUNCTION UNDER NORMAL AND DEGENERATIVE CONDITIONS, Journal of neurochemistry, 69, 1997, pp. 188-188
AXONALLY-TRANSPORTED NEUROFILAMENT PROTEINS EXIST IN MULTIPLE SOLUBILITY STATES IN MOUSE OPTIC PATHWAY AND NB2A D1 CELLS/
Authors: JUNG C WHEELER E DAHL D SHEA TB
Citation: C. Jung et al., AXONALLY-TRANSPORTED NEUROFILAMENT PROTEINS EXIST IN MULTIPLE SOLUBILITY STATES IN MOUSE OPTIC PATHWAY AND NB2A D1 CELLS/, Journal of neurochemistry, 69, 1997, pp. 189-189
TAU-PHOSPHORYLATION IS REGULATED BY DIVERGENT AND CONVERGENT SIGNAL-TRANSDUCTION PATHWAYS
Authors: EKINCI F PUNDREDDY S WHEELER E SHEA TB
Citation: F. Ekinci et al., TAU-PHOSPHORYLATION IS REGULATED BY DIVERGENT AND CONVERGENT SIGNAL-TRANSDUCTION PATHWAYS, Journal of neurochemistry, 69, 1997, pp. 208-208
INDUCTION OF LYSOSOMAL ABNORMALITIES AND TAU-HYPERPHOSPHORYLATION IN HUMAN NEUROBLASTOMA-CELLS BY COLCHICINE AND OKADAIC ACID - EVIDENCE THAT MICROTUBULE DISRUPTION CONTRIBUTES TO ALZHEIMER NEURODEGENERATION
Authors: SHEA TB
Citation: Tb. Shea, INDUCTION OF LYSOSOMAL ABNORMALITIES AND TAU-HYPERPHOSPHORYLATION IN HUMAN NEUROBLASTOMA-CELLS BY COLCHICINE AND OKADAIC ACID - EVIDENCE THAT MICROTUBULE DISRUPTION CONTRIBUTES TO ALZHEIMER NEURODEGENERATION, Neuroscience research communications, 19(1), 1996, pp. 27-36
THE INFLUENCE OF SURFACE PROTEASE ACTIVITY ON NEURITOGENESIS AND NEURITE RETRACTION IS DEPENDENT UPON MODE OF NEURITE INDUCTION - INHIBITION OF THROMBIN-MEDIATED SURFACE PROTEASE ACTIVITY IS CRUCIAL FOR NEURITE OUTGROWTH BUT NOT FOR MAINTENANCE OF EXISTING NEURITES
Authors: SHEA TB
Citation: Tb. Shea, THE INFLUENCE OF SURFACE PROTEASE ACTIVITY ON NEURITOGENESIS AND NEURITE RETRACTION IS DEPENDENT UPON MODE OF NEURITE INDUCTION - INHIBITION OF THROMBIN-MEDIATED SURFACE PROTEASE ACTIVITY IS CRUCIAL FOR NEURITE OUTGROWTH BUT NOT FOR MAINTENANCE OF EXISTING NEURITES, Neuroscience research communications, 19(1), 1996, pp. 57-60
REGULATION OF NEURONAL DIFFERENTIATION BY THE ALPHA-ISOFORM AND EPSILON-ISOFORM OF PROTEIN-KINASE-C
Authors: BOYCE JJ ENNULAT DJ LELI U SHEA TB
Citation: Jj. Boyce et al., REGULATION OF NEURONAL DIFFERENTIATION BY THE ALPHA-ISOFORM AND EPSILON-ISOFORM OF PROTEIN-KINASE-C, Neuroscience research communications, 18(3), 1996, pp. 195-201
INDUCTION OF NEURITE OUTGROWTH IN NEUROBLASTOMA-CELLS BY ASTROGLIAL-SECRETED FACTORS IS MEDIATED BY PROTEIN-KINASE-C
Authors: SHEA TB HUSAIN T
Citation: Tb. Shea et T. Husain, INDUCTION OF NEURITE OUTGROWTH IN NEUROBLASTOMA-CELLS BY ASTROGLIAL-SECRETED FACTORS IS MEDIATED BY PROTEIN-KINASE-C, Neuroscience research communications, 18(1), 1996, pp. 57-61
ASTROGLIAL-SECRETED FACTORS INDUCE THE ELABORATION OF UNSTABLE AXONALNEURITES BY NB2A D1 NEUROBLASTOMA-CELLS - CORRELATION WITH DEFICIENCIES IN MAP1B AND PHOSPHO-ISOFORMS OF THE HIGH-MOLECULAR-WEIGHT NEUROFILAMENT SUBUNIT/
Authors: SHEA TB HUSAIN T
Citation: Tb. Shea et T. Husain, ASTROGLIAL-SECRETED FACTORS INDUCE THE ELABORATION OF UNSTABLE AXONALNEURITES BY NB2A D1 NEUROBLASTOMA-CELLS - CORRELATION WITH DEFICIENCIES IN MAP1B AND PHOSPHO-ISOFORMS OF THE HIGH-MOLECULAR-WEIGHT NEUROFILAMENT SUBUNIT/, Neuroscience research communications, 18(1), 1996, pp. 63-70
ROLE OF VIMENTIN IN EARLY STAGES OF NEURITOGENESIS IN CULTURED HIPPOCAMPAL-NEURONS
Authors: BOYNE LJ FISCHER I SHEA TB
Citation: Lj. Boyne et al., ROLE OF VIMENTIN IN EARLY STAGES OF NEURITOGENESIS IN CULTURED HIPPOCAMPAL-NEURONS, International journal of developmental neuroscience, 14(6), 1996, pp. 739-748
SPECTRIN-ACTIN INTERACTION IS REQUIRED FOR NEURITE EXTENSION IN NE 2ADL NEUROBLASTOMA-CELLS/
Authors: SIHAG RK SHEA TB WANG FS
Citation: Rk. Sihag et al., SPECTRIN-ACTIN INTERACTION IS REQUIRED FOR NEURITE EXTENSION IN NE 2ADL NEUROBLASTOMA-CELLS/, Journal of neuroscience research, 44(5), 1996, pp. 430-437
CALCIUM INFLUX INTO HUMAN NEUROBLASTOMA-CELLS INDUCES ALZ-50 IMMUNOREACTIVITY - INVOLVEMENT OF CALPAIN-MEDIATED HYDROLYSIS OF PROTEIN-KINASE-C
Authors: SHEA TB SPENCER MJ BEERMANN ML CRESSMAN CM NIXON RA
Citation: Tb. Shea et al., CALCIUM INFLUX INTO HUMAN NEUROBLASTOMA-CELLS INDUCES ALZ-50 IMMUNOREACTIVITY - INVOLVEMENT OF CALPAIN-MEDIATED HYDROLYSIS OF PROTEIN-KINASE-C, Journal of neurochemistry, 66(4), 1996, pp. 1539-1549
SITE-SPECIFIC SYNERGISM BETWEEN PKC AND MAP KINASE IN TAU-HYPERPHOSPHORYLATION IN INTACT SH-SY-5Y HUMAN NEUROBLASTOMA
Authors: CRESSMAN CM BOYCE JB WHEELER E CHANG M SHEA TB
Citation: Cm. Cressman et al., SITE-SPECIFIC SYNERGISM BETWEEN PKC AND MAP KINASE IN TAU-HYPERPHOSPHORYLATION IN INTACT SH-SY-5Y HUMAN NEUROBLASTOMA, Journal of neurochemistry, 66, 1996, pp. 62-62
DIFFERENTIAL-EFFECTS OF BRYOSTATIN AND PHORBOL ESTERS ON NEUROBLASTOMA-CELLS - UNIQUE ROLE OF PROTEIN-KINASE C-EPSILON
Authors: EKINCI FJ BOYCE JB SHEA TB
Citation: Fj. Ekinci et al., DIFFERENTIAL-EFFECTS OF BRYOSTATIN AND PHORBOL ESTERS ON NEUROBLASTOMA-CELLS - UNIQUE ROLE OF PROTEIN-KINASE C-EPSILON, Journal of neurochemistry, 66, 1996, pp. 62-62
PREFERENTIAL TRANSLOCATION OF HMW-TAU ISOFORMS INTO NEWLY-ELABORATED NEURITES IN HUMAN NEUROBLASTOMA
Authors: SHEA TB CRESSMAN CM FISCHER I
Citation: Tb. Shea et al., PREFERENTIAL TRANSLOCATION OF HMW-TAU ISOFORMS INTO NEWLY-ELABORATED NEURITES IN HUMAN NEUROBLASTOMA, Journal of neurochemistry, 66, 1996, pp. 63-63
ROLE OF VIMENTIN IN EARLY STAGES OF NEURITOGENSIS IN CULTURED HIPPOCAMPAL-NEURONS
Authors: BOYNE LJ FISCHER I SHEA TB
Citation: Lj. Boyne et al., ROLE OF VIMENTIN IN EARLY STAGES OF NEURITOGENSIS IN CULTURED HIPPOCAMPAL-NEURONS, Journal of neurochemistry, 66, 1996, pp. 65-65
PHOSPHATASE INHIBITION IN HUMAN NEUROBLASTOMA-CELLS ALTERS TAU ANTIGENICITY AND RENDERS IT INCOMPETENT TO ASSOCIATE WITH EXOGENOUS MICROTUBULES
Authors: SHEA TB FISCHER I
Citation: Tb. Shea et I. Fischer, PHOSPHATASE INHIBITION IN HUMAN NEUROBLASTOMA-CELLS ALTERS TAU ANTIGENICITY AND RENDERS IT INCOMPETENT TO ASSOCIATE WITH EXOGENOUS MICROTUBULES, FEBS letters, 380(1-2), 1996, pp. 63-67
PKC-EPSILON AND PKC-ALPHA REGULATE TAU LEVELS AND PHOSPHORYLATION STATE
Authors: BOYCE JJ ENNULAT DJ NEVE R SHEA TB
Citation: Jj. Boyce et al., PKC-EPSILON AND PKC-ALPHA REGULATE TAU LEVELS AND PHOSPHORYLATION STATE, Molecular biology of the cell, 6, 1995, pp. 214-214
SPECTRIN-ACTIN INTERACTION IS REQUIRED FOR NEURITE EXTENSION IN NB 2AD1 NEUROBLASTOMA-CELLS/
Authors: SIHAG RK SHEA TB WANG FS
Citation: Rk. Sihag et al., SPECTRIN-ACTIN INTERACTION IS REQUIRED FOR NEURITE EXTENSION IN NB 2AD1 NEUROBLASTOMA-CELLS/, Molecular biology of the cell, 6, 1995, pp. 2162-2162
NEUROFILAMENT TRANSLOCATION INTO AXONAL NEURITES IS DISTINCT FROM INCORPORATION INTO THE CYTOSKELETON
Authors: SHEA TB WHEELER E WANG FS
Citation: Tb. Shea et al., NEUROFILAMENT TRANSLOCATION INTO AXONAL NEURITES IS DISTINCT FROM INCORPORATION INTO THE CYTOSKELETON, Molecular biology of the cell, 6, 1995, pp. 2177-2177
INHIBITION OF PROTEOLYSIS ENHANCES ALUMINUM-INDUCED PERIKARYAL NEUROFILAMENT ACCUMULATION BUT DOES NOT ENHANCE TAU ACCUMULATION
Authors: SHEA TB HUSAIN T
Citation: Tb. Shea et T. Husain, INHIBITION OF PROTEOLYSIS ENHANCES ALUMINUM-INDUCED PERIKARYAL NEUROFILAMENT ACCUMULATION BUT DOES NOT ENHANCE TAU ACCUMULATION, Molecular and chemical neuropathology, 26(3), 1995, pp. 195-212
ALUMINUM TREATMENT OF INTACT NEUROBLASTOMA-CELLS ALTERS NEUROFILAMENTSUBUNIT PHOSPHORYLATION, SOLUBILITY, AND PROTEOLYSIS
Authors: SHEA TB BEERMANN ML NIXON RA
Citation: Tb. Shea et al., ALUMINUM TREATMENT OF INTACT NEUROBLASTOMA-CELLS ALTERS NEUROFILAMENTSUBUNIT PHOSPHORYLATION, SOLUBILITY, AND PROTEOLYSIS, Molecular and chemical neuropathology, 26(1), 1995, pp. 1-14
CALCIUM MODULATES ALUMINUM NEUROTOXICITY AND INTERACTION WITH NEUROFILAMENTS
Authors: SHEA TB
Citation: Tb. Shea, CALCIUM MODULATES ALUMINUM NEUROTOXICITY AND INTERACTION WITH NEUROFILAMENTS, Molecular and chemical neuropathology, 24(2-3), 1995, pp. 151-163
RELATIVE SUSCEPTIBILITY OF CYTOSKELETON-ASSOCIATED AND SOLUBLE NEUROFILAMENT SUBUNITS TO ALUMINUM EXPOSURE IN INTACT-CELLS - A POSSIBLE MECHANISM FOR REDUCTION OF NEUROFILAMENT AXONAL-TRANSPORT DURING ALUMINUMNEUROTOXICITY
Authors: SHEA TB BEERMANN ML WANG FS
Citation: Tb. Shea et al., RELATIVE SUSCEPTIBILITY OF CYTOSKELETON-ASSOCIATED AND SOLUBLE NEUROFILAMENT SUBUNITS TO ALUMINUM EXPOSURE IN INTACT-CELLS - A POSSIBLE MECHANISM FOR REDUCTION OF NEUROFILAMENT AXONAL-TRANSPORT DURING ALUMINUMNEUROTOXICITY, Molecular and chemical neuropathology, 24(2-3), 1995, pp. 203-219
CALCIUM INFLUX RECRUITS AN ADDITIONAL CLASS OF KINASES TO HYPERPHOSPHORYLATE-TAU
Authors: SHEA TB KLINGER EP CRESSMAN CM
Citation: Tb. Shea et al., CALCIUM INFLUX RECRUITS AN ADDITIONAL CLASS OF KINASES TO HYPERPHOSPHORYLATE-TAU, NeuroReport, 6(10), 1995, pp. 1437-1440
ALTERATION IN TAU ANTIGENICITY AND ELECTROPHORETIC MIGRATION BY PKC-ALPHA UNDER CELL-FREE CONDITIONS
Authors: CRESSMAN CM MERCKEN MM SHEA TB
Citation: Cm. Cressman et al., ALTERATION IN TAU ANTIGENICITY AND ELECTROPHORETIC MIGRATION BY PKC-ALPHA UNDER CELL-FREE CONDITIONS, Neuroscience research communications, 17(2), 1995, pp. 61-64
Risultati: 1-25 | 26-50 | 51-75 | 76-79