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Results: 1-12 |
Results: 12

Authors: CRAIN SM SHEN KF
Citation: Sm. Crain et Kf. Shen, MODULATION OF OPIOID ANALGESIA, TOLERANCE AND DEPENDENCE BY G(S)-COUPLED, GM1 GANGLIOSIDE-REGULATED OPIOID RECEPTOR FUNCTIONS, Trends in pharmacological sciences, 19(9), 1998, pp. 358-365

Authors: SHEN KF CRAIN SM
Citation: Kf. Shen et Sm. Crain, ULTRA-LOW DOSES OF NALTREXONE OR ETORPHINE INCREASE MORPHINES ANTINOCICEPTIVE POTENCY AND ATTENUATE TOLERANCE DEPENDENCE IN MICE/, Brain research, 757(2), 1997, pp. 176-190

Authors: CRAIN SM SHEN KF
Citation: Sm. Crain et Kf. Shen, MODULATORY EFFECTS OF GS-COUPLED EXCITATORY OPIOID RECEPTOR FUNCTIONSON OPIOID ANALGESIA, TOLERANCE, AND DEPENDENCE, Neurochemical research, 21(11), 1996, pp. 1347-1351

Authors: KITANIDIS PK SHEN KF
Citation: Pk. Kitanidis et Kf. Shen, GEOSTATISTICAL INTERPOLATION OF CHEMICAL CONCENTRATION, Advances in water resources, 19(6), 1996, pp. 369-378

Authors: CRAIN SM SHEN KF
Citation: Sm. Crain et Kf. Shen, ETORPHINE ELICITS ANOMALOUS EXCITATORY OPIOID EFFECTS ON SENSORY NEURONS TREATED WITH GM1 GANGLIOSIDE OR PERTUSSIS TOXIN IN CONTRAST TO ITSPOTENT INHIBITORY EFFECTS ON NAIVE OR CHRONIC MORPHINE-TREATED CELLS, Brain research, 741(1-2), 1996, pp. 275-283

Authors: CRAIN SM SHEN KF
Citation: Sm. Crain et Kf. Shen, ULTRA-LOW CONCENTRATIONS OF NALOXONE SELECTIVELY ANTAGONIZE EXCITATORY EFFECTS OF MORPHINE ON SENSORY NEURONS, THEREBY INCREASING ITS ANTINOCICEPTIVE POTENCY AND ATTENUATING TOLERANCE DEPENDENCE DURING CHRONICCOTREATMENT, Proceedings of the National Academy of Sciences of the United Statesof America, 92(23), 1995, pp. 10540-10544

Authors: SHEN KF CRAIN SM
Citation: Kf. Shen et Sm. Crain, BIPHALIN, AN ENKEPHALIN ANALOG WITH UNEXPECTEDLY HIGH ANTINOCICEPTIVEPOTENCY AND LOW DEPENDENCE LIABILITY IN-VIVO, SELECTIVELY ANTAGONIZESEXCITATORY OPIOID RECEPTOR FUNCTIONS OF SENSORY NEURONS IN CULTURE, Brain research, 701(1-2), 1995, pp. 158-166

Authors: CRAIN SM SHEN KF
Citation: Sm. Crain et Kf. Shen, CHRONIC MORPHINE-TREATED SENSORY GANGLION NEURONS REMAIN SUPERSENSITIVE TO THE EXCITATORY EFFECTS OF NALOXONE FOR MONTHS AFTER RETURN TO NORMAL CULTURE-MEDIUM - AN IN-VITRO MODEL OF PROTRACTED OPIOID DEPENDENCE, Brain research, 694(1-2), 1995, pp. 103-110

Authors: SHEN KF CRAIN SM
Citation: Kf. Shen et Sm. Crain, SPECIFIC N-TERMINUS OR C-TERMINUS MODIFIED DYNORPHIN AND BETA-ENDORPHIN PEPTIDES CAN SELECTIVELY BLOCK EXCITATORY OPIOID RECEPTOR FUNCTIONSIN SENSORY NEURONS AND UNMASK POTENT INHIBITORY EFFECTS OF OPIOID AGONISTS, Brain research, 673(1), 1995, pp. 30-38

Authors: SHEN KF CRAIN SM
Citation: Kf. Shen et Sm. Crain, NERVE GROWTH-FACTOR RAPIDLY PROLONGS THE ACTION-POTENTIAL OF MATURE SENSORY GANGLION NEURONS IN CULTURE, AND THIS EFFECT REQUIRES ACTIVATION OF GS-COUPLED EXCITATORY KAPPA-OPIOID RECEPTORS ON THESE CELLS, The Journal of neuroscience, 14(9), 1994, pp. 5570-5579

Authors: SHEN KF CRAIN SM
Citation: Kf. Shen et Sm. Crain, ETORPHINE ELICITS UNIQUE DOSE-DEPENDENT INHIBITORY EFFECTS ON THE ACTION-POTENTIAL DURATION OF SENSORY NEURONS EVEN AT LOW CONCENTRATIONS WHERE OTHER OPIOIDS ELICIT EXCITATORY EFFECTS, Regulatory peptides, 1994, pp. 190000079-190000080

Authors: SHEN KF CRAIN SM
Citation: Kf. Shen et Sm. Crain, ANTAGONISTS AT EXCITATORY OPIOID RECEPTORS ON SENSORY NEURONS IN CULTURE INCREASE POTENCY AND SPECIFICITY OF OPIATE ANALGESICS AND ATTENUATE DEVELOPMENT OF TOLERANCE DEPENDENCE/, Brain research, 636(2), 1994, pp. 286-297
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