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Results: 1-7 |
Results: 7
BAK CAN ACCELERATE CHEMOTHERAPY-INDUCED CELL-DEATH INDEPENDENTLY OF ITS HETERODIMERIZATION WITH BCL-X-L AND BCL-2
Authors: SIMONIAN PL GRILLOT DAM NUNEZ G
Citation: Pl. Simonian et al., BAK CAN ACCELERATE CHEMOTHERAPY-INDUCED CELL-DEATH INDEPENDENTLY OF ITS HETERODIMERIZATION WITH BCL-X-L AND BCL-2, Oncogene, 15(15), 1997, pp. 1871-1875
BCL-2 AND BCL-XL CAN DIFFERENTIALLY BLOCK CHEMOTHERAPY-INDUCED CELL-DEATH
Authors: SIMONIAN PL GRILLOT DAM NUNEZ G
Citation: Pl. Simonian et al., BCL-2 AND BCL-XL CAN DIFFERENTIALLY BLOCK CHEMOTHERAPY-INDUCED CELL-DEATH, Blood, 90(3), 1997, pp. 1208-1216
ISOLATION AND PROPAGATION OF THE KAPOSIS-SARCOMA (ICS)-ASSOCIATED HERPESVIRUS (HHV-8) FROM CULTURED KS TUMOR-CELLS GROWN UNDER NOVEL CONDITIONS
Authors: FOREMAN KE FRIBORG J WOFFENDIN C SIMONIAN PL POLVERINI PJ NABEL GJ NICKOLOFF BJ
Citation: Ke. Foreman et al., ISOLATION AND PROPAGATION OF THE KAPOSIS-SARCOMA (ICS)-ASSOCIATED HERPESVIRUS (HHV-8) FROM CULTURED KS TUMOR-CELLS GROWN UNDER NOVEL CONDITIONS, Journal of investigative dermatology, 106(4), 1996, pp. 58-58
BAX HOMODIMERIZATION IS NOT REQUIRED FOR BAX TO ACCELERATE CHEMOTHERAPY-INDUCED CELL-DEATH
Authors: SIMONIAN PL GRILLOT DAM ANDREWS DW LEBER B NUNEZ G
Citation: Pl. Simonian et al., BAX HOMODIMERIZATION IS NOT REQUIRED FOR BAX TO ACCELERATE CHEMOTHERAPY-INDUCED CELL-DEATH, The Journal of biological chemistry, 271(50), 1996, pp. 32073-32077
PAX CAN ANTAGONIZE BCL-X(L) DURING ETOPOSIDE AND CISPLATIN-INDUCED CELL-DEATH INDEPENDENTLY OF ITS HETERODIMERIZATION WITH BCL-X(L)
Authors: SIMONIAN PL GRILLOT DAM MERINO R NUNEZ G
Citation: Pl. Simonian et al., PAX CAN ANTAGONIZE BCL-X(L) DURING ETOPOSIDE AND CISPLATIN-INDUCED CELL-DEATH INDEPENDENTLY OF ITS HETERODIMERIZATION WITH BCL-X(L), The Journal of biological chemistry, 271(37), 1996, pp. 22764-22772
KAPOSIS-SARCOMA TUMOR-CELLS PREFERENTIALLY EXPRESS BCL-X(L)
Authors: FOREMAN KE WRONESMITH T BOISE LH THOMPSON CB POLVERINI PJ SIMONIAN PL NUNEZ G NICKOLOFF BJ
Citation: Ke. Foreman et al., KAPOSIS-SARCOMA TUMOR-CELLS PREFERENTIALLY EXPRESS BCL-X(L), The American journal of pathology, 149(3), 1996, pp. 795-803
MODULATION OF ANTI-IGM-INDUCED B-CELL APOPTOSIS BY BCL-X(L) AND CD40 IN WEHI-231 CELLS - DISSOCIATION FROM CELL-CYCLE ARREST AND DEPENDENCEON THE AVIDITY OF THE ANTIBODY-IGM RECEPTOR INTERACTION
Authors: MERINO R GRILLOT DAM SIMONIAN PL MUTHUKKUMAR S FANSLOW WC BONDADA S NUNEZ G
Citation: R. Merino et al., MODULATION OF ANTI-IGM-INDUCED B-CELL APOPTOSIS BY BCL-X(L) AND CD40 IN WEHI-231 CELLS - DISSOCIATION FROM CELL-CYCLE ARREST AND DEPENDENCEON THE AVIDITY OF THE ANTIBODY-IGM RECEPTOR INTERACTION, The Journal of immunology, 155(8), 1995, pp. 3830-3838
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