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WANG HL
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FUKUDOME T
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BRENGMAN JM
GRIGGS RC
SINE SM
ENGEL AG
Citation: M. Milone et al., MODE SWITCHING KINETICS PRODUCED BY A NATURALLY-OCCURRING MUTATION INTHE CYTOPLASMIC LOOP OF THE HUMAN ACETYLCHOLINE-RECEPTOR EPSILON-SUBUNIT, Neuron, 20(3), 1998, pp. 575-588
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MARCHOT P
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Citation: N. Sugiyama et al., RESIDUES AT THE SUBUNIT INTERFACES OF THE NICOTINIC ACETYLCHOLINE-RECEPTOR THAT CONTRIBUTE TO ALPHA-CONOTOXIN M1 BINDING, Molecular pharmacology, 53(4), 1998, pp. 787-794
Citation: Pa. Quiram et Sm. Sine, IDENTIFICATION OF RESIDUES IN THE NEURONAL ALPHA(7) ACETYLCHOLINE-RECEPTOR THAT CONFER SELECTIVITY FOR CONOTOXIN IMI, The Journal of biological chemistry, 273(18), 1998, pp. 11001-11006
Citation: Pa. Quiram et Sm. Sine, STRUCTURAL ELEMENTS IN ALPHA-CONOTOXIN IMI ESSENTIAL FOR BINDING TO NEURONAL ALPHA(7) RECEPTORS, The Journal of biological chemistry, 273(18), 1998, pp. 11007-11011
Citation: Rj. Prince et Sm. Sine, EPIBATIDINE BINDS WITH UNIQUE SITE AND STATE SELECTIVITY TO MUSCLE NICOTINIC ACETYLCHOLINE-RECEPTORS, The Journal of biological chemistry, 273(14), 1998, pp. 7843-7849
Citation: Rj. Prince et Sm. Sine, EPIBATIDINE ACTIVATES MUSCLE ACETYLCHOLINE-RECEPTORS WITH UNIQUE SITESELECTIVITY, Biophysical journal, 75(4), 1998, pp. 1817-1827
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OHNO K
QUIRAM PA
MILONE M
WANG HL
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Citation: K. Ohno et al., CONGENITAL MYASTHENIC SYNDROMES DUE TO HETEROALLELIC NONSENSE MISSENSE MUTATIONS IN THE ACETYLCHOLINE-RECEPTOR EPSILON-SUBUNIT GENE - IDENTIFICATION AND FUNCTIONAL-CHARACTERIZATION OF 6 NEW MUTATIONS/, Human molecular genetics, 6(5), 1997, pp. 753-766
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Citation: M. Milone et al., SLOW-CHANNEL MYASTHENIC SYNDROME CAUSED BY ENHANCED ACTIVATION, DESENSITIZATION, AND AGONIST BINDING-AFFINITY ATTRIBUTABLE TO MUTATION IN THE M2 DOMAIN OF THE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNIT, The Journal of neuroscience, 17(15), 1997, pp. 5651-5665
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Citation: K. Ohno et al., CONGENITAL MYASTHENIC SYNDROMES (CMS) DUE TO HETEROALLELIC NONSENSE MISSENSE MUTATIONS IN THE ACETYLCHOLINE-RECEPTOR (ACHR) EPSILON-SUBUNITGENE - REPORT OF 5 NEW MUTATIONS/, Neurology, 48(3), 1997, pp. 1003-1003
Authors:
WANG HL
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Citation: Hl. Wang et al., MUTATION IN THE M1 DOMAIN OF THE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNITDECREASES THE RATE OF AGONIST DISSOCIATION (VOL 109, PG 758, 1997), The Journal of general physiology, 110(2), 1997, pp. 215-215
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Citation: Hl. Wang et al., MUTATION IN THE M1 DOMAIN OF THE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNITDECREASES THE RATE OF AGONIST DISSOCIATION, The Journal of general physiology, 109(6), 1997, pp. 757-766
Citation: N. Bren et Sm. Sine, IDENTIFICATION OF RESIDUES IN THE ADULT NICOTINIC ACETYLCHOLINE-RECEPTOR THAT CONFER SELECTIVITY FOR CURARIFORM ANTAGONISTS, The Journal of biological chemistry, 272(49), 1997, pp. 30793-30798
Citation: Sm. Sine, IDENTIFICATION OF EQUIVALENT RESIDUES IN THE GAMMA-SUBUNIT, DELTA-SUBUNIT, AND EPSILON-SUBUNIT OF THE NICOTINIC RECEPTOR THAT CONTRIBUTE TOALPHA-BUNGAROTOXIN BINDING, The Journal of biological chemistry, 272(38), 1997, pp. 23521-23527
Citation: I. Tsigelny et al., A MODEL OF THE NICOTINIC RECEPTOR EXTRACELLULAR DOMAIN BASED ON SEQUENCE IDENTITY AND RESIDUE LOCATION, Biophysical journal, 73(1), 1997, pp. 52-66
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ENGEL AG
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MILONE M
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Citation: Ag. Engel et al., NEW MUTATIONS IN ACETYLCHOLINE-RECEPTOR SUBUNIT GENES REVEAL HETEROGENEITY IN THE SLOW-CHANNEL CONGENITAL MYASTHENIC SYNDROME, Human molecular genetics, 5(9), 1996, pp. 1217-1227
Authors:
OHNO K
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PRUITT JN
SINE SM
ENGEL AG
Citation: K. Ohno et al., CONGENITAL MYASTHENIC SYNDROME CAUSED BY DECREASED AGONIST BINDING-AFFINITY DUE TO A MUTATION IN THE ACETYLCHOLINE-RECEPTOR EPSILON-SUBUNIT, Neuron, 17(1), 1996, pp. 157-170
Authors:
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KAWANISHI C
MARCHOT P
MOLLES BE
BOYD AE
SINE SM
TAYLOR PW
Citation: N. Sugiyama et al., SUBSTITUENTS IN THE ALPHA-SUBUNIT OF NICOTINIC ACETYLCHOLINE-RECEPTOR(NACHR) WHICH DICTATE ALPHA-CONOTOXIN M1 (ALPHA-CTX-M1) BINDING, The FASEB journal, 10(3), 1996, pp. 3972-3972
Authors:
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OHNO K
WANG HL
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SINE SM
ENGEL AG
Citation: M. Milone et al., NOVEL SLOW-CHANNEL SYNDROME DUE TO MUTATION IN THE ACETYLCHOLINE-RECEPTOR (ACHR) ALPHA-SUBUNIT WITH INCREASED CONDUCTANCE, NANOMOLAR AFFINITY FOR ACETYLCHOLINE, AND PROLONGED OPEN DURATIONS OF THE ACHR CHANNEL, Annals of neurology, 40(6), 1996, pp. 9-9
Authors:
ENGEL AG
OHNO K
BOUZAT C
SINE SM
GRIGGS RC
Citation: Ag. Engel et al., END-PLATE ACETYLCHOLINE-RECEPTOR DEFICIENCY DUE TO NONSENSE MUTATIONSIN THE EPSILON-SUBUNIT, Annals of neurology, 40(5), 1996, pp. 810-817