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Citation: Wf. Graier et al., SUBMAXIMAL STIMULATION OF PORCINE ENDOTHELIAL-CELLS CAUSES FOCAL CA2-MEMBRANE( ELEVATION BENEATH THE CELL), Journal of physiology, 506(1), 1998, pp. 109-125
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Citation: Br. Wamhoff et al., EVIDENCE FOR SARCOPLASMIC-RETICULUM REGULATION OF NUCLEAR FUNCTION INA MODEL FOR CORONARY-ARTERY DISEASE, The Journal of general physiology, 112(1), 1998, pp. 122-122
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Citation: Cl. Heaps et al., EFFECTS OF EXERCISE TRAINING ON SMOOTH-MUSCLE RELAXATION AND MYOPLASMIC FREE CA2-M) RESPONSES OF CORONARY-ARTERIES DISTAL TO CHRONIC OCCLUSION( (CA), The FASEB journal, 11(3), 1997, pp. 2779-2779
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Citation: Cl. Heaps et al., IMPAIRED CAMP-MEDIATED VASORELAXATION OF PORCINE COLLATERAL-DEPENDENTARTERIES DISTAL TO CHRONIC CORONARY-OCCLUSION - EFFECTS OF EXERCISE TRAINING, Circulation, 96(8), 1997, pp. 1737-1737
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Citation: Ja. Rapps et al., MECHANISMS OF ALTERED CONTRACTILE RESPONSES TO VASOPRESSIN AND ENDOTHELIN IN CANINE CORONARY COLLATERAL ARTERIES, Circulation, 95(1), 1997, pp. 231-239
Citation: Dl. Lee et M. Sturek, ENDOTHELIN CAUSES PROLONGED POTENTIATION OF DEPOLARIZATION-INDUCED CALCIUM INFLUX, BUT DESENSITIZATION OF CALCIUM-RELEASE IN CORONARY SMOOTH-MUSCLE, The FASEB journal, 10(3), 1996, pp. 388-388
Citation: Dk. Bowles et M. Sturek, ACTIVATION OF A LARGE-CONDUCTANCE, CA2-ACTIVATED K+ CHANNEL BY ADENOSINE IN PORCINE CORONARY ARTERIAL SMOOTH-MUSCLE(), The FASEB journal, 10(3), 1996, pp. 402-402
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