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Results: 1-25 |
Results: 25
Defining a common region of deletion at 13q21 in human cancers
Authors: Chen, CS Brabham, WW Stultz, BG Frierson, HF Barrett, JC Sawyers, CL Isaacs, JT Dong, JT
Citation: Cs. Chen et al., Defining a common region of deletion at 13q21 in human cancers, GENE CHROM, 31(4), 2001, pp. 333-344
Cancer treatment in the STI571 era: What will change?
Authors: Sawyers, CL
Citation: Cl. Sawyers, Cancer treatment in the STI571 era: What will change?, J CL ONCOL, 19(18), 2001, pp. 13S-16S
Implications of signal transduction inhibition for the treatment of chronic myeloid leukemia
Authors: Sawyers, CL
Citation: Cl. Sawyers, Implications of signal transduction inhibition for the treatment of chronic myeloid leukemia, SEM HEMATOL, 38(3), 2001, pp. 1-2
Molecular studies in chronic myeloid leukemia patients treated with tyrosine kinase inhibitors
Authors: Sawyers, CL
Citation: Cl. Sawyers, Molecular studies in chronic myeloid leukemia patients treated with tyrosine kinase inhibitors, SEM HEMATOL, 38(3), 2001, pp. 15-21
Research on resistance to cancer drug Gleevec
Authors: Sawyers, CL
Citation: Cl. Sawyers, Research on resistance to cancer drug Gleevec, SCIENCE, 294(5548), 2001, pp. 1834-1834
Roots of clinical resistance to STI-571 cancer therapy - Response
Authors: Gorre, M Shah, N Ellwood, K Nicoll, J Sawyers, CL
Citation: M. Gorre et al., Roots of clinical resistance to STI-571 cancer therapy - Response, SCIENCE, 293(5538), 2001, pp. NIL_3-NIL_5
Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification
Authors: Gorre, ME Mohammed, M Ellwood, K Hsu, N Paquette, R Rao, PN Sawyers, CL
Citation: Me. Gorre et al., Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification, SCIENCE, 293(5531), 2001, pp. 876-880
Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinasein chronic myeloid leukemia.
Authors: Druker, BJ Talpaz, M Resta, DJ Peng, B Buchdunger, E Ford, JM Lydon, NB Kantarjian, H Capdeville, R Ohno-Jones, S Sawyers, CL
Citation: Bj. Druker et al., Efficacy and safety of a specific inhibitor of the BCR-ABL tyrosine kinasein chronic myeloid leukemia., N ENG J MED, 344(14), 2001, pp. 1031-1037
Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia withthe philadelphia chromosome.
Authors: Druker, BJ Sawyers, CL Kantarjian, H Resta, DJ Reese, SF Ford, JM Capdeville, R Talpaz, M
Citation: Bj. Druker et al., Activity of a specific inhibitor of the BCR-ABL tyrosine kinase in the blast crisis of chronic myeloid leukemia and acute lymphoblastic leukemia withthe philadelphia chromosome., N ENG J MED, 344(14), 2001, pp. 1038-1042
Enhanced sensitivity of PTEN-deficient tumors to inhibition of FRAP/mTOR
Authors: Neshat, MS Mellinghoff, IK Tran, C Stiles, B Thomas, G Petersen, R Frost, P Gibbons, JJ Wu, H Sawyers, CL
Citation: Ms. Neshat et al., Enhanced sensitivity of PTEN-deficient tumors to inhibition of FRAP/mTOR, P NAS US, 98(18), 2001, pp. 10314-10319
In vivo progression of LAPC-9 and LNCaP prostate cancer models to androgenindependence is associated with increased expression of insulin-like growth factor I (IGF-I) and IGF-I receptor (IGF-IR)
Authors: Nickerson, T Chang, F Lorimer, D Smeekens, SP Sawyers, CL Pollak, M
Citation: T. Nickerson et al., In vivo progression of LAPC-9 and LNCaP prostate cancer models to androgenindependence is associated with increased expression of insulin-like growth factor I (IGF-I) and IGF-I receptor (IGF-IR), CANCER RES, 61(16), 2001, pp. 6276-6280
Xenograft models and the molecular biology of human prostate cancer
Authors: Reiter, RE Sawyers, CL
Citation: Re. Reiter et Cl. Sawyers, Xenograft models and the molecular biology of human prostate cancer, CONT CANC R, 2001, pp. 163-174
The survival function of the Bcr-Abl oncogene is mediated by Bad-dependentand -independent pathways: Roles for phosphatidylinositol 3-kinase and Raf
Authors: Neshat, MS Raitano, AB Wang, HG Reed, JC Sawyers, CL
Citation: Ms. Neshat et al., The survival function of the Bcr-Abl oncogene is mediated by Bad-dependentand -independent pathways: Roles for phosphatidylinositol 3-kinase and Raf, MOL CELL B, 20(4), 2000, pp. 1179-1186
Evidence for regulation of the PTEN tumor suppressor by a membrane-localized multi-PDZ domain containing scaffold protein MAGI-2
Authors: Wu, XY Hepner, K Castelino-Prabhu, S Do, D Kaye, MB Yuan, XJ Wood, J Ross, C Sawyers, CL Whang, YE
Citation: Xy. Wu et al., Evidence for regulation of the PTEN tumor suppressor by a membrane-localized multi-PDZ domain containing scaffold protein MAGI-2, P NAS US, 97(8), 2000, pp. 4233-4238
c-Abl is required for development and optimal cell proliferation in the context of p53 deficiency
Authors: Whang, YE Tran, C Henderson, C Syljuasen, RG Rozengurt, N McBride, WH Sawyers, CL
Citation: Ye. Whang et al., c-Abl is required for development and optimal cell proliferation in the context of p53 deficiency, P NAS US, 97(10), 2000, pp. 5486-5491
Tyrosine kinase inhibitors in chronic myeloid leukemia
Authors: Sawyers, CL Druker, B
Citation: Cl. Sawyers et B. Druker, Tyrosine kinase inhibitors in chronic myeloid leukemia, CA J SCI AM, 5(2), 1999, pp. 63-69
A mechanism for hormone-independent prostate cancer through modulation of androgen receptor signaling by the HER-2/neu tyrosine kinase
Authors: Craft, N Shostak, Y Carey, M Sawyers, CL
Citation: N. Craft et al., A mechanism for hormone-independent prostate cancer through modulation of androgen receptor signaling by the HER-2/neu tyrosine kinase, NAT MED, 5(3), 1999, pp. 280-285
Mitogen-activated protein kinase kinase kinase 1 activates androgen receptor-dependent transcription and apoptosis in prostate cancer
Authors: Abreu-Martin, MT Chari, A Palladino, AA Craft, NA Sawyers, CL
Citation: Mt. Abreu-martin et al., Mitogen-activated protein kinase kinase kinase 1 activates androgen receptor-dependent transcription and apoptosis in prostate cancer, MOL CELL B, 19(7), 1999, pp. 5143-5154
Chronic myeloid leukemia - Reply
Authors: Sawyers, CL
Citation: Cl. Sawyers, Chronic myeloid leukemia - Reply, N ENG J MED, 341(10), 1999, pp. 765-765
Chronic myeloid leukemia
Authors: Sawyers, CL
Citation: Cl. Sawyers, Chronic myeloid leukemia, N ENG J MED, 340(17), 1999, pp. 1330-1340
Evidence for clonal outgrowth of androgen-independent prostate cancer cells from androgen-dependent tumors through a two-step process
Authors: Craft, N Chhor, C Tran, C Belldegrun, A DeKernion, J Witte, ON Said, J Reiter, RE Sawyers, CL
Citation: N. Craft et al., Evidence for clonal outgrowth of androgen-independent prostate cancer cells from androgen-dependent tumors through a two-step process, CANCER RES, 59(19), 1999, pp. 5030-5036
C/EBP alpha bypasses granulocyte colony-stimulating factor signals to rapidly induce PU.1 gene expression, stimulate granulocytic differentiation, and limit proliferation in 32D cl3 myeloblasts
Authors: Wang, XP Scott, E Sawyers, CL Friedman, AD
Citation: Xp. Wang et al., C/EBP alpha bypasses granulocyte colony-stimulating factor signals to rapidly induce PU.1 gene expression, stimulate granulocytic differentiation, and limit proliferation in 32D cl3 myeloblasts, BLOOD, 94(2), 1999, pp. 560-571
Mechanistic concepts in androgen-dependence of prostate cancer
Authors: Craft, N Sawyers, CL
Citation: N. Craft et Cl. Sawyers, Mechanistic concepts in androgen-dependence of prostate cancer, CANC METAST, 17(4), 1998, pp. 421-427
Molecular abnormalities in myeloid leukemias and myelodysplastic syndromes
Authors: Sawyers, CL
Citation: Cl. Sawyers, Molecular abnormalities in myeloid leukemias and myelodysplastic syndromes, LEUK RES, 22(12), 1998, pp. 1113-1122
The PTEN/MMAC1 tumor suppressor phosphatase functions as a negative regulator of the phosphoinositide 3-kinase/Akt pathway
Authors: Wu, XY Senechal, K Neshat, MS Whang, YE Sawyers, CL
Citation: Xy. Wu et al., The PTEN/MMAC1 tumor suppressor phosphatase functions as a negative regulator of the phosphoinositide 3-kinase/Akt pathway, P NAS US, 95(26), 1998, pp. 15587-15591
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