Citation: Dv. Serreze et Eh. Leiter, Genes and cellular requirements for autoimmune diabetes susceptibility in nonobese diabetic mice, CURR DIR AU, 4, 2001, pp. 31-67
Authors:
Hamilton-Williams, EE
Serreze, DV
Charlton, B
Johnson, EA
Marron, MP
Mullbacher, A
Slattery, RM
Citation: Ee. Hamilton-williams et al., Transgenic rescue implicates beta(2)-microglobulin as a diabetes susceptibility gene in nonobese diabetic (NOD) mice, P NAS US, 98(20), 2001, pp. 11533-11538
Authors:
Mathews, CE
Graser, RT
Savinov, A
Serreze, DV
Leiter, EH
Citation: Ce. Mathews et al., Unusual resistance of ALR/Lt mouse beta cells to autoimmune destruction: Role for beta cell-expressed resistance determinants, P NAS US, 98(1), 2001, pp. 235-240
Authors:
Johnson, EA
Silveira, P
Chapman, HD
Leiter, EH
Serreze, DV
Citation: Ea. Johnson et al., Inhibition of autoimmune diabetes in nonobese diabetic mice by transgenic restoration of H2-E MHC class II expression: Additive, but unequal, involvement of multiple APC subtypes, J IMMUNOL, 167(4), 2001, pp. 2404-2410
Authors:
Tisch, R
Wang, B
Weaver, DJ
Liu, S
Bui, T
Arthos, J
Serreze, DV
Citation: R. Tisch et al., Antigen-specific mediated suppression of beta cell autoimmunity by plasmidDNA vaccination, J IMMUNOL, 166(3), 2001, pp. 2122-2132
Authors:
Serreze, DV
Chapman, HD
Post, CM
Johnson, EA
Suarez-Pinzon, WL
Rabinovitch, A
Citation: Dv. Serreze et al., Th1 to Th2 cytokine shifts in nonobese diabetic mice: Sometimes an outcome, rather than the cause, of diabetes resistance elicited by immunostimulation, J IMMUNOL, 166(2), 2001, pp. 1352-1359
Authors:
Serreze, DV
Johnson, EA
Chapman, HD
Graser, RT
Marron, MP
DiLorenzo, TP
Silveira, P
Yoshimura, Y
Nathenson, SG
Joyce, S
Citation: Dv. Serreze et al., Autoreactive diabetogenic T-cells in NOD mice can efficiently expand from a greatly reduced precursor pool, DIABETES, 50(9), 2001, pp. 1992-2000
Citation: Ppl. Chiu et al., Development and function of diabetogenic T-cells in B-cell-deficient nonobese diabetic mice, DIABETES, 50(4), 2001, pp. 763-770
Authors:
Graser, RT
DiLorenzo, TP
Wang, FM
Christianson, GJ
Chapman, HD
Roopenian, DC
Nathenson, SG
Serreze, DV
Citation: Rt. Graser et al., Identification of a CD8 T cell that can independently mediate autoimmune diabetes development in the complete absence of CD4 T cell helper functions, J IMMUNOL, 164(7), 2000, pp. 3913-3918
Authors:
Serreze, DV
Ottendorfer, EW
Ellis, TM
Gauntt, CJ
Atkinson, MA
Citation: Dv. Serreze et al., Acceleration of type 1 diabetes by a coxsackievirus infection requires a preexisting critical mass of autoreactive T-cells in pancreatic islets, DIABETES, 49(5), 2000, pp. 708-711
Authors:
Serreze, DV
Post, CM
Chapman, HD
Johnson, EA
Lu, BF
Rothman, PB
Citation: Dv. Serreze et al., Interferon-gamma receptor signaling is dispensable in the development of autoimmune type 1 diabetes in NOD mice, DIABETES, 49(12), 2000, pp. 2007-2011
Authors:
Mathews, CE
Graser, RT
Serreze, DV
Leiter, EH
Citation: Ce. Mathews et al., Reevaluation of the major histocompatibility complex genes of the NOD-progenitor CTS/Shi strain, DIABETES, 49(1), 2000, pp. 131-134
Authors:
Graser, RT
Mathews, CE
Leiter, EH
Serreze, DV
Citation: Rt. Graser et al., MHC characterization of ALR and ALS mice: respective similarities to the NOD and NON strains, IMMUNOGENET, 49(7-8), 1999, pp. 722-726
Authors:
Trembleau, S
Penna, G
Gregori, S
Chapman, HD
Serreze, DV
Magram, J
Adorini, L
Citation: S. Trembleau et al., Pancreas-infiltrating Th1 cells and diabetes develop in IL-12-deficient nonobese diabetic mice, J IMMUNOL, 163(5), 1999, pp. 2960-2968
Citation: R. Tisch et al., Induction of glutamic acid decarboxylase 65-specific Th2 cells and suppression of autoimmune diabetes at late stages of disease is epitope dependent, J IMMUNOL, 163(3), 1999, pp. 1178-1187
Authors:
Markees, TG
Serreze, DV
Phillips, NE
Sorli, CH
Gordon, EJ
Shultz, LD
Noelle, RJ
Woda, BA
Greiner, DL
Mordes, JP
Rossini, AA
Citation: Tg. Markees et al., NOD mice have a generalized defect in their response to transplantation tolerance induction, DIABETES, 48(5), 1999, pp. 967-974