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Citation: T. Saetre et al., AMINOETHYL-ISOTHIOUREA, A SELECTIVE INHIBITOR OF INDUCIBLE NITRIC-OXIDE SYNTHASE ACTIVITY, IMPROVES LIVER CIRCULATION AND OXYGEN-METABOLISMIN A PORCINE MODEL OF ENDOTOXEMIA, Shock, 9(2), 1998, pp. 109-115
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Citation: Y. Hattori et al., INDUCTION OF NITRIC-OXIDE AND TETRAHYDROBIOPTERIN SYNTHESIS BY LIPOTEICHOIC ACID FROM STAPHYLOCOCCUS-AUREUS IN VASCULAR SMOOTH-MUSCLE CELLS, Journal of vascular research, 35(2), 1998, pp. 104-108
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Citation: Km. Kengatharan et al., MECHANISM OF GRAM-POSITIVE SHOCK - IDENTIFICATION OF PEPTIDOGLYCAN AND LIPOTEICHOIC ACID MOIETIES ESSENTIAL IN THE INDUCTION OF NITRIC-OXIDE SYNTHASE, SHOCK, AND MULTIPLE ORGAN FAILURE, The Journal of experimental medicine, 188(2), 1998, pp. 305-315
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Citation: K. Zacharowski et al., 2 DISTINCT EP3-RECEPTOR AGONISTS REDUCE MYOCARDIAL INFARCT SIZE - ROLE OF MITOCHONDRIAL ATP-SENSITIVE POTASSIUM CHANNELS, Circulation, 98(17), 1998, pp. 3982-3982
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Citation: M. Leach et al., DECLINE IN THE EXPRESSION OF COPPER ZINC SUPEROXIDE-DISMUTASE IN THE KIDNEY OF RATS WITH ENDOTOXIC-SHOCK - EFFECTS OF THE SUPEROXIDE ANION-RADICAL SCAVENGER, TEMPOL, ON ORGAN INJURY/, British Journal of Pharmacology, 125(4), 1998, pp. 817-825
Citation: J. Bowes et C. Thiemermann, THE NO DONOR SPM-5186 REDUCES INFARCT SIZE IN A RABBIT MODEL OF MYOCARDIAL-ISCHEMIA AND REPERFUSION, British Journal of Pharmacology, 124, 1998, pp. 15-15
Citation: K. Zacharowski et al., THE SELECTIVE EP3-PROSTANOID RECEPTOR AGONIST, ONO-AE-248, REDUCES INFARCT SIZE IN A RAT MODEL OF MYOCARDIAL-ISCHEMIA AND REPERFUSION, British Journal of Pharmacology, 124, 1998, pp. 104-104
Citation: J. Bowes et al., THE PARS INHIBITORS 3-AMINOBENZAMIDE, NICOTINAMIDE AND 1,5-DIHYDROXYISOQUINOLINE - DO NOT SCAVENGE SUPEROXIDE ANIONS IN-VITRO, British Journal of Pharmacology, 124, 1998, pp. 105-105
Citation: J. Bowes et al., INHIBITORS OF THE ACTIVITY OF POLY (ADP-RIBOSE) SYNTHETASE REDUCE THECELL-DEATH CAUSED BY HYDROGEN-PEROXIDE IN HUMAN CARDIAC MYOBLASTS, British Journal of Pharmacology, 124(8), 1998, pp. 1760-1766
Citation: J. Bowes et C. Thiemermann, EFFECTS OF INHIBITORS OF THE ACTIVITY OF POLY (ADP-RIBOSE) SYNTHETASEON THE LIVER-INJURY CAUSED BY ISCHEMIA-REPERFUSION - A COMPARISON WITH RADICAL SCAVENGERS, British Journal of Pharmacology, 124(6), 1998, pp. 1254-1260
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Citation: M. Leach et al., EFFECTS OF INHIBITORS OF THE ACTIVITY OF CYCLO-OXYGENASE-2 ON THE HYPOTENSION AND MULTIPLE ORGAN DYSFUNCTION CAUSED BY ENDOTOXIN - A COMPARISON WITH DEXAMETHASONE, British Journal of Pharmacology, 124(3), 1998, pp. 586-592
Authors:
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Citation: G. Wray et al., THE SELECTIVE INHIBITOR OF INDUCIBLE NITRIC-OXIDE SYNTHASE, 1400W, PREVENTS THE CIRCULATORY FAILURE BUT NOT THE ORGAN INJURY CAUSED BY ENDOTOXIN IN THE RAT, British Journal of Pharmacology, 123, 1998, pp. 5-5
Citation: J. Bowes et al., INHIBITION OF THE ACTIVITY OF POLY(ADP-RIBOSE) SYNTHETASE REDUCES CELL INJURY CAUSED BY HYDROGEN-PEROXIDE IN RAT CARDIAC MYOBLASTS, British Journal of Pharmacology, 123, 1998, pp. 93-93
Citation: J. Bowes et C. Thiemermann, COMPARISON OF THE EFFECTS OF INHIBITORS OF POLY(ADP-RIBOSE) SYNTHETASE AND RADICAL SCAVENGERS IN A RAT MODEL OF HEPATIC ISCHEMIA AND, British Journal of Pharmacology, 123, 1998, pp. 282-282
Citation: M. Leach et al., EFFECTS OF SC-58635, A SELECTIVE INHIBITOR OF CYCLOOXYGENASE-2 ON HEMODYNAMICS AND ORGAN FUNCTION OF RATS WITH ENDOTOXEMIA, British Journal of Pharmacology, 123, 1998, pp. 311-311
Citation: H. Ruetten et C. Thiemermann, INTERLEUKIN-13 IS A MORE POTENT INHIBITOR OF THE EXPRESSION OF INDUCIBLE NITRIC-OXIDE SYNTHASE IN SMOOTH-MUSCLE CELLS THAN IN MACROPHAGES -A COMPARISON WITH INTERLEUKIN-4 AND INTERLEUKIN-10, Shock, 8(6), 1997, pp. 409-414
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Citation: Cgm. Millar et al., PRESERVATION OF RENAL MITOCHONDRIAL-FUNCTION IN ENDOTOXEMIC ACUTE-RENAL-FAILURE, Journal of the American Society of Nephrology, 8, 1997, pp. 2755-2755
Citation: H. Ruetten et C. Thiemermann, COMBINATION IMMUNOTHERAPY WHICH NEUTRALIZES THE EFFECTS OF TNF-ALPHA AND IL-1-BETA ATTENUATES THE CIRCULATORY FAILURE AND MULTIPLE ORGAN DYSFUNCTION CAUSED BY ENDOTOXIN IN THE RAT, Journal of Physiology and Pharmacology, 48(4), 1997, pp. 605-621
Citation: H. Ruetten et C. Thiemermann, ENDOTHELIN-1 STIMULATES THE BIOSYNTHESIS OF TUMOR-NECROSIS-FACTOR IN MACROPHAGES - ET-RECEPTORS, SIGNAL-TRANSDUCTION AND INHIBITION BY DEXAMETHASONE, Journal of Physiology and Pharmacology, 48(4), 1997, pp. 675-688