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Results: 1-12 |
Results: 12
Enhanced TRAIL sensitivity by p53 overexpression in human cancer but not normal cell lines
Authors: Kim, K Takimoto, R Dicker, DT Chen, YH Gazitt, Y El-Deiry, WS
Citation: K. Kim et al., Enhanced TRAIL sensitivity by p53 overexpression in human cancer but not normal cell lines, INT J ONCOL, 18(2), 2001, pp. 241-247
hADA3 is required for p53 activity
Authors: Wang, T Kobayashi, T Takimoto, R Denes, AE Snyder, EL el-Deiry, WS Brachmann, RK
Citation: T. Wang et al., hADA3 is required for p53 activity, EMBO J, 20(22), 2001, pp. 6404-6413
DNA replication blockade impairs p53-transactivation
Authors: Takimoto, R El-Deiry, WS
Citation: R. Takimoto et Ws. El-deiry, DNA replication blockade impairs p53-transactivation, P NAS US, 98(3), 2001, pp. 781-783
The bile acid glycochenodeoxycholate induces TRAIL-receptor 2/DR5 expression and apoptosis
Authors: Higuchi, H Bronk, SF Takikawa, Y Werneburg, N Takimoto, R El-Deiry, W Gores, GJ
Citation: H. Higuchi et al., The bile acid glycochenodeoxycholate induces TRAIL-receptor 2/DR5 expression and apoptosis, J BIOL CHEM, 276(42), 2001, pp. 38610-38618
Glutathione S-transferase-pi overexpression is closely associated with K-ras mutation during human colon carcinogenesis
Authors: Miyanishi, K Takayama, T Ohi, M Hayashi, T Nobuoka, A Nakajima, T Takimoto, R Kogawa, K Kato, J Sakamaki, S Niitsu, Y
Citation: K. Miyanishi et al., Glutathione S-transferase-pi overexpression is closely associated with K-ras mutation during human colon carcinogenesis, GASTROENTY, 121(4), 2001, pp. 865-874
Analysis of K-ras, APC, and beta-catenin in aberrant crypt foci in sporadic adenoma, cancer, and familial adenomatous polyposis
Authors: Takayama, T Ohi, M Hayashi, T Miyanishi, K Nobuoka, A Nakajima, T Satoh, T Takimoto, R Kato, J Sakamaki, S Niitsu, Y
Citation: T. Takayama et al., Analysis of K-ras, APC, and beta-catenin in aberrant crypt foci in sporadic adenoma, cancer, and familial adenomatous polyposis, GASTROENTY, 121(3), 2001, pp. 599-611
A mutation, in the iron-responsive element of H ferritin mRNA, causing autosomal dominant iron overload
Authors: Kato, J Fujikawa, K Kanda, M Fukuda, N Sasaki, K Takayama, T Kobune, M Takada, K Takimoto, R Hamada, H Ikeda, T Niitsu, Y
Citation: J. Kato et al., A mutation, in the iron-responsive element of H ferritin mRNA, causing autosomal dominant iron overload, AM J HU GEN, 69(1), 2001, pp. 191-197
Wild-type p53 transactivates the KILLER/DR5 gene through an intronic sequence-specific DNA-binding site
Authors: Takimoto, R El-Deiry, WS
Citation: R. Takimoto et Ws. El-deiry, Wild-type p53 transactivates the KILLER/DR5 gene through an intronic sequence-specific DNA-binding site, ONCOGENE, 19(14), 2000, pp. 1735-1743
Nasal natural killer cell lymphoma presenting as lethal midline granuloma
Authors: Yamazaki, M Kakuta, M Takimoto, R Murayama, N Takamori, K
Citation: M. Yamazaki et al., Nasal natural killer cell lymphoma presenting as lethal midline granuloma, INT J DERM, 39(12), 2000, pp. 931-934
Sauer's manual of skin diseases (vol 142, pg 1268, 2000)
Authors: Hase, T Shinta, K Murase, T Tokimitsu, I Hattori, M Takimoto, R Tsuboi, R Ogawa, H
Citation: T. Hase et al., Sauer's manual of skin diseases (vol 142, pg 1268, 2000), BR J DERM, 143(2), 2000, pp. 470-470
Histological increase in inflammatory infiltrate in sun-exposed skin of female subjects: the possible involvement of matrix metalloproteinase-1 produced by inflammatory infiltrate on collagen degradation
Authors: Hase, T Shinta, K Murase, T Tokimitsu, I Hattori, M Takimoto, R Tsuboi, R Ogawa, H
Citation: T. Hase et al., Histological increase in inflammatory infiltrate in sun-exposed skin of female subjects: the possible involvement of matrix metalloproteinase-1 produced by inflammatory infiltrate on collagen degradation, BR J DERM, 142(2), 2000, pp. 267-273
Transforming growth factor-beta 1 (TGF-beta 1) induces thrombopoietin frombone marrow stromal cells, which stimulates the expression of TGF-beta receptor on megakaryocytes and, in turn, renders them susceptible to suppression by TGF-beta itself with high specificity
Authors: Sakamaki, S Hirayama, Y Matsunaga, T Kuroda, H Kusakabe, T Akiyama, T Konuma, Y Sasaki, K Tsuji, N Okamoto, T Kobune, M Kogawa, K Kato, J Takimoto, R Koyama, R Niitsu, Y
Citation: S. Sakamaki et al., Transforming growth factor-beta 1 (TGF-beta 1) induces thrombopoietin frombone marrow stromal cells, which stimulates the expression of TGF-beta receptor on megakaryocytes and, in turn, renders them susceptible to suppression by TGF-beta itself with high specificity, BLOOD, 94(6), 1999, pp. 1961-1970
Risultati: 1-12 |