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Citation: N. Unno et al., HYPERPERMEABILITY OF INTESTINAL EPITHELIAL MONOLAYERS IS INDUCED BY NO - EFFECT OF LOW EXTRACELLULAR PH, American journal of physiology: Gastrointestinal and liver physiology, 35(5), 1997, pp. 923-934
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Citation: Mj. Menconi et al., ACIDOSIS INDUCES HYPERPERMEABILITY IN CACO-2BBE CULTURED INTESTINAL EPITHELIAL MONOLAYERS, American journal of physiology: Gastrointestinal and liver physiology, 35(5), 1997, pp. 1007-1021
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Citation: N. Unno et al., AN EVALUATION OF THE SYSTEM TO CONTROL BLOOD-FLOW IN MAINTAINING GOATFETUSES ON ARTERIOVENOUS EXTRACORPOREAL MEMBRANE-OXYGENATION - A NOVEL-APPROACH TO THE DEVELOPMENT OF AN ARTIFICIAL PLACENTA, Artificial organs, 21(12), 1997, pp. 1239-1246
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Citation: N. Unno et al., ACIDIC CONDITIONS AMELIORATE BOTH ADENOSINE-TRIPHOSPHATE DEPLETION AND THE DEVELOPMENT OF HYPERPERMEABILITY IN CULTURED CACO-2(BBE) ENTEROCYTIC MONOLAYERS SUBJECTED TO METABOLIC INHIBITION, Surgery, 121(6), 1997, pp. 668-680
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Citation: N. Unno et al., INHIBITION OF INDUCIBLE NITRIC-OXIDE SYNTHASE AMELIORATES ENDOTOXIN-INDUCED GUT MUCOSAL BARRIER DYSFUNCTION IN RATS, Gastroenterology, 113(4), 1997, pp. 1246-1257
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Citation: N. Unno et al., CHANGES IN ADRENOCORTICOTROPIN AND CORTISOL RESPONSIVENESS AFTER REPEATED PARTIAL UMBILICAL-CORD OCCLUSIONS IN THE LATE-GESTATION OVINE FETUS, Endocrinology, 138(1), 1997, pp. 259-263
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Citation: N. Unno et al., HYPERPERMEABILITY AND ATP DEPLETION INDUCED BY CHRONIC HYPOXIA OR GLYCOLYTIC INHIBITION IN CACO-2BBE MONOLAYERS, American journal of physiology: Gastrointestinal and liver physiology, 33(6), 1996, pp. 1010-1021
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Citation: N. Unno et al., NITRIC-OXIDE (NO)-INDUCED DERANGEMENTS IN THE BARRIER FUNCTION OF INTESTINAL EPITHELIAL MONOLAYERS - EFFECT OF LOW PHO, Gastroenterology, 110(4), 1996, pp. 370-370
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Citation: Al. Salzman et al., NITRIC-OXIDE DILATES TIGHT JUNCTIONS AND DEPLETES ATP IN CULTURED CACO-2BBE INTESTINAL EPITHELIAL MONOLAYERS, American journal of physiology: Gastrointestinal and liver physiology, 31(2), 1995, pp. 361-373
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