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Results: 1-12 |
Results: 12
N-TERMINUS OF SOS1 RAS EXCHANGE FACTOR - CRITICAL ROLES FOR THE DBL AND PLECKSTRIN HOMOLOGY DOMAINS
Authors: QIAN XL VASS WC PAPAGEORGE AG ANBORGH PH LOWY DR
Citation: Xl. Qian et al., N-TERMINUS OF SOS1 RAS EXCHANGE FACTOR - CRITICAL ROLES FOR THE DBL AND PLECKSTRIN HOMOLOGY DOMAINS, Molecular and cellular biology, 18(2), 1998, pp. 771-778
THE RAS-SPECIFIC EXCHANGE FACTORS MOUSE SOS1 (MSOS1) AND MSOS2 ARE REGULATED DIFFERENTLY - MSOS2 CONTAINS UBIQUITINATION SIGNALS ABSENT IN MSOS1
Authors: NIELSEN KH PAPAGEORGE AG VASS WC WILLUMSEN BM LOWY DR
Citation: Kh. Nielsen et al., THE RAS-SPECIFIC EXCHANGE FACTORS MOUSE SOS1 (MSOS1) AND MSOS2 ARE REGULATED DIFFERENTLY - MSOS2 CONTAINS UBIQUITINATION SIGNALS ABSENT IN MSOS1, Molecular and cellular biology, 17(12), 1997, pp. 7132-7138
A CRITICAL ROLE FOR THE DBL AND PH DOMAINS OF THE RAS-SPECIFIC EXCHANGE FACTOR SOS
Authors: QIAN XL VASS WC LOWY DR
Citation: Xl. Qian et al., A CRITICAL ROLE FOR THE DBL AND PH DOMAINS OF THE RAS-SPECIFIC EXCHANGE FACTOR SOS, Journal of investigative dermatology, 108(4), 1997, pp. 46-46
COLOCALIZATION OF THE TSC2 PRODUCT TUBERIN WITH ITS TARGET RAP1 IN THE GOLGI-APPARATUS
Authors: WIENECKE R MAIZE JC SHOARINEJAD F VASS WC REED J BONIFACINO JS RESAU JH DEGUNZBURG J YEUNG RS DECLUE JE
Citation: R. Wienecke et al., COLOCALIZATION OF THE TSC2 PRODUCT TUBERIN WITH ITS TARGET RAP1 IN THE GOLGI-APPARATUS, Oncogene, 13(5), 1996, pp. 913-923
REGULATION OF RAS ACTIVITY IN CELLULAR SIGNALING
Authors: LOWY DR DECLUE JE VASS WC PAPAGEORGE AG NIELSEN KH WILLUMSEN BM
Citation: Dr. Lowy et al., REGULATION OF RAS ACTIVITY IN CELLULAR SIGNALING, AIDS research and human retroviruses, 11, 1995, pp. 64-64
SHC IS CONSTITUTIVELY ACTIVATED BY A TRANSFORMING EGF RECEPTOR MUTANTWITH A POINT MUTATION IN THE TRANSMEMBRANE DOMAIN
Authors: BEGUINOT L VASS WC MILOSO M
Citation: L. Beguinot et al., SHC IS CONSTITUTIVELY ACTIVATED BY A TRANSFORMING EGF RECEPTOR MUTANTWITH A POINT MUTATION IN THE TRANSMEMBRANE DOMAIN, Journal of cellular biochemistry, 1995, pp. 11-11
SHC AND GRB-2 ARE CONSTITUTIVELY ACTIVATED BY AN EPIDERMAL GROWTH-FACTOR RECEPTOR WITH A POINT MUTATION IN THE TRANSMEMBRANE DOMAIN
Authors: MILOSO M MAZZOTTI M VASS WC BEGUINOT L
Citation: M. Miloso et al., SHC AND GRB-2 ARE CONSTITUTIVELY ACTIVATED BY AN EPIDERMAL GROWTH-FACTOR RECEPTOR WITH A POINT MUTATION IN THE TRANSMEMBRANE DOMAIN, The Journal of biological chemistry, 270(33), 1995, pp. 19557-19562
NEUROFIBROMIN CAN INHIBIT RAS-DEPENDENT GROWTH BY A MECHANISM INDEPENDENT OF ITS GTPASE-ACCELERATING FUNCTION
Authors: JOHNSON MR DECLUE JE FELZMANN S VASS WC XU GF WHITE R LOWY DR
Citation: Mr. Johnson et al., NEUROFIBROMIN CAN INHIBIT RAS-DEPENDENT GROWTH BY A MECHANISM INDEPENDENT OF ITS GTPASE-ACCELERATING FUNCTION, Molecular and cellular biology, 14(1), 1994, pp. 641-645
REGULATED AND CONSTITUTIVE ACTIVITY BY CDC25(MM) (GRF), A RAS-SPECIFIC EXCHANGE FACTOR
Authors: CEN H PAPAGEORGE AG VASS WC ZHANG K LOWY DR
Citation: H. Cen et al., REGULATED AND CONSTITUTIVE ACTIVITY BY CDC25(MM) (GRF), A RAS-SPECIFIC EXCHANGE FACTOR, Molecular and cellular biology, 13(12), 1993, pp. 7718-7724
FUNCTIONAL-ROLE OF GTPASE-ACTIVATING PROTEIN IN CELL-TRANSFORMATION BY PP60(V-SRC)
Authors: DECLUE JE VASS WC JOHNSON MR STACEY DW LOWY DR
Citation: Je. Declue et al., FUNCTIONAL-ROLE OF GTPASE-ACTIVATING PROTEIN IN CELL-TRANSFORMATION BY PP60(V-SRC), Molecular and cellular biology, 13(11), 1993, pp. 6799-6809
TRANSFORMATION-SPECIFIC INTERACTION OF THE BOVINE PAPILLOMAVIRUS E5 ONCOPROTEIN WITH THE PLATELET-DERIVED GROWTH-FACTOR RECEPTOR TRANSMEMBRANE DOMAIN AND THE EPIDERMAL GROWTH-FACTOR RECEPTOR CYTOPLASMIC DOMAIN
Authors: COHEN BD GOLDSTEIN DJ RUTLEDGE L VASS WC LOWY DR SCHLEGEL R SCHILLER JT
Citation: Bd. Cohen et al., TRANSFORMATION-SPECIFIC INTERACTION OF THE BOVINE PAPILLOMAVIRUS E5 ONCOPROTEIN WITH THE PLATELET-DERIVED GROWTH-FACTOR RECEPTOR TRANSMEMBRANE DOMAIN AND THE EPIDERMAL GROWTH-FACTOR RECEPTOR CYTOPLASMIC DOMAIN, Journal of virology, 67(9), 1993, pp. 5303-5311
NEUROFIBROMIN CAN INHIBIT RAS-DEPENDENT GROWTH BY A MECHANISM INDEPENDENT OF ITS GTPASE ACCELERATING FUNCTION
Authors: JOHNSON MR DECLUE JE FELZMANN S VASS WC XU GF WHITE R LOWY DR
Citation: Mr. Johnson et al., NEUROFIBROMIN CAN INHIBIT RAS-DEPENDENT GROWTH BY A MECHANISM INDEPENDENT OF ITS GTPASE ACCELERATING FUNCTION, Clinical research, 41(3), 1993, pp. 10000607-10000607
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