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Results: 1-25 | 26-50 | 51-75 | 76-77
Results: 26-50/77
GRADUAL REPERFUSION REDUCES INFARCT SIZE AND ENDOTHELIAL INJURY BUT AUGMENTS NEUTROPHIL ACCUMULATION
Authors: SATO H JORDAN JE ZHAO ZQ SARVOTHAM SS VINTENJOHANSEN J
Citation: H. Sato et al., GRADUAL REPERFUSION REDUCES INFARCT SIZE AND ENDOTHELIAL INJURY BUT AUGMENTS NEUTROPHIL ACCUMULATION, The Annals of thoracic surgery, 64(4), 1997, pp. 1099-1107
CLORICROMENE REDUCES MYOCARDIAL INFARCT SIZE IN RABBITS WHEN ADMINISTERED DURING THE EARLY REPERFUSION PERIOD
Authors: ZVARA DA GALASKA HJ CASTELLANO VP VINTENJOHANSEN J ROYSTER RL WILLIAMS MW MURPHY BA KIM EJ
Citation: Da. Zvara et al., CLORICROMENE REDUCES MYOCARDIAL INFARCT SIZE IN RABBITS WHEN ADMINISTERED DURING THE EARLY REPERFUSION PERIOD, Anesthesia and analgesia, 84(2), 1997, pp. 266-270
CLORICROMENE REDUCES INFARCT SIZE AFTER AN ISCHEMIC-REPERFUSION INJURY IN THE CANINE MYOCARDIUM
Authors: GROBAN L ZVARA DA DEAL DD FLYE CW VINTENJOHANSEN J
Citation: L. Groban et al., CLORICROMENE REDUCES INFARCT SIZE AFTER AN ISCHEMIC-REPERFUSION INJURY IN THE CANINE MYOCARDIUM, Anesthesia and analgesia, 84, 1997, pp. 80-80
CLORICROMENE INHIBITS PMN ADHERENCE TO CANINE CORONARY-ARTERY ENDOTHELIUM
Authors: ZVARA DA DEAL DD WILLIAMS MW VINTENJOHANSEN J GROBAN L
Citation: Da. Zvara et al., CLORICROMENE INHIBITS PMN ADHERENCE TO CANINE CORONARY-ARTERY ENDOTHELIUM, Anesthesia and analgesia, 84, 1997, pp. 142-142
ADENOSINE (ADO) INHIBITS UNACTIVATED NEUTROPHIL (PMN) ADHERENCE TO THROMBIN-STIMULATED CORONARY VASCULAR ENDOTHELIUM (EC) BY A P-SELECTIN MECHANISM
Authors: WILLIAMS MW JORDAN JE FERNANDEZ AZ ZHAO ZQ VINTENJOHANSEN J
Citation: Mw. Williams et al., ADENOSINE (ADO) INHIBITS UNACTIVATED NEUTROPHIL (PMN) ADHERENCE TO THROMBIN-STIMULATED CORONARY VASCULAR ENDOTHELIUM (EC) BY A P-SELECTIN MECHANISM, The FASEB journal, 10(3), 1996, pp. 1613-1613
EXOGENOUS NO INHIBITS BASAL NO RELEASE FROM VASCULAR ENDOTHELIUM IN-VITRO AND IN-VIVO
Authors: MA XL LOPEZ BL CHRISTOPHER TA BIRENBAUM DS VINTENJOHANSEN J
Citation: Xl. Ma et al., EXOGENOUS NO INHIBITS BASAL NO RELEASE FROM VASCULAR ENDOTHELIUM IN-VITRO AND IN-VIVO, American journal of physiology. Heart and circulatory physiology, 40(5), 1996, pp. 2045-2051
ADENOSINE A(2)-RECEPTOR ACTIVATION INHIBITS NEUTROPHIL-MEDIATED INJURY TO CORONARY ENDOTHELIUM
Authors: ZHAO ZQ SATO H WILLIAMS MW FERNANDEZ AZ VINTENJOHANSEN J
Citation: Zq. Zhao et al., ADENOSINE A(2)-RECEPTOR ACTIVATION INHIBITS NEUTROPHIL-MEDIATED INJURY TO CORONARY ENDOTHELIUM, American journal of physiology. Heart and circulatory physiology, 40(4), 1996, pp. 1456-1464
MYOCARDIAL PROTECTION IN NORMAL AND HYPOXICALLY STRESSED NEONATAL HEARTS - THE SUPERIORITY OF HYPOCALCEMIC VERSUS NORMOCALCEMIC BLOOD CARDIOPLEGIA - DISCUSSION
Authors: KENNEDY JH",BOLLING,"VINTENJOHANSEN J
Citation: J. Kennedy Jh",bolling,"vintenjohansen, MYOCARDIAL PROTECTION IN NORMAL AND HYPOXICALLY STRESSED NEONATAL HEARTS - THE SUPERIORITY OF HYPOCALCEMIC VERSUS NORMOCALCEMIC BLOOD CARDIOPLEGIA - DISCUSSION, Journal of thoracic and cardiovascular surgery, 112(5), 1996, pp. 1200-1201
CARDIOPROTECTION BY ADENOSINE - IS IT A QUESTION OF EFFECTIVE DOSE, TIMING, OR THE TARGET COMPARTMENT
Authors: VINTENJOHANSEN J HAMMON JW
Citation: J. Vintenjohansen et Jw. Hammon, CARDIOPROTECTION BY ADENOSINE - IS IT A QUESTION OF EFFECTIVE DOSE, TIMING, OR THE TARGET COMPARTMENT, Journal of thoracic and cardiovascular surgery, 112(1), 1996, pp. 202-203
DEPRESSION OF CARDIAC-FUNCTION AFTER DEEP HYPOTHERMIC CIRCULATORY ARREST IN DEEPLY ANESTHETIZED NEONATAL LAMBS
Authors: VELVIS H HINES MH KLOPFENSTEIN HS BERRY DD VINTENJOHANSEN J
Citation: H. Velvis et al., DEPRESSION OF CARDIAC-FUNCTION AFTER DEEP HYPOTHERMIC CIRCULATORY ARREST IN DEEPLY ANESTHETIZED NEONATAL LAMBS, Journal of thoracic and cardiovascular surgery, 111(2), 1996, pp. 359-366
VOLUME EXPANSION INCREASES RIGHT-VENTRICULAR INFARCT SIZE IN DOGS BY REDUCING COLLATERAL PERFUSION
Authors: JOHNSTON WE LIN CY FEERICK AE SPRAY B VINTENJOHANSEN J
Citation: We. Johnston et al., VOLUME EXPANSION INCREASES RIGHT-VENTRICULAR INFARCT SIZE IN DOGS BY REDUCING COLLATERAL PERFUSION, Chest, 109(2), 1996, pp. 494-503
ADENOSINE REDUCES NEUTROPHIL ADHERENCE TO STIMULATED ENDOTHELIUM BY INHIBITING STIMULUS INITIATION
Authors: WILLIAMS MW FERNANDEZ AZ JORDAN JE ZHAO ZQ RILEY RD VINTENJOHANSEN J
Citation: Mw. Williams et al., ADENOSINE REDUCES NEUTROPHIL ADHERENCE TO STIMULATED ENDOTHELIUM BY INHIBITING STIMULUS INITIATION, Circulation, 94(8), 1996, pp. 2856-2856
L-ARGININE INHIBITS NEUTROPHIL ADHERENCE AND CORONARY-ARTERY DYSFUNCTION
Authors: SATO H ZHAO ZQ VINTENJOHANSEN J
Citation: H. Sato et al., L-ARGININE INHIBITS NEUTROPHIL ADHERENCE AND CORONARY-ARTERY DYSFUNCTION, Cardiovascular Research, 31(1), 1996, pp. 63-72
INTRAVASCULAR ADENOSINE AT REPERFUSION REDUCES INFARCT SIZE AND NEUTROPHIL ADHERENCE
Authors: TODD J ZHAO ZQ WILLIAMS MW SATO H VANWYLEN DGL VINTENJOHANSEN J
Citation: J. Todd et al., INTRAVASCULAR ADENOSINE AT REPERFUSION REDUCES INFARCT SIZE AND NEUTROPHIL ADHERENCE, The Annals of thoracic surgery, 62(5), 1996, pp. 1364-1372
CLORICROMENE IMPROVES MYOCARDIAL BLOOD-FLOW IN A CANINE MODEL OF ISCHEMIC-REPERFUSION INJURY
Authors: GROBAN I ZVARA DA DEAL DD VERNON JC VINTENJOHANSEN J
Citation: I. Groban et al., CLORICROMENE IMPROVES MYOCARDIAL BLOOD-FLOW IN A CANINE MODEL OF ISCHEMIC-REPERFUSION INJURY, Anesthesiology, 85(3A), 1996, pp. 602-602
REDUCTION IN INFARCT SIZE BUT NOT NEUTROPHIL ACCUMULATION BY CONTROLLED HYDRODYNAMICS OF REPERFUSION
Authors: VINTENJOHANSEN J ZHAO ZQ SATO H JORDAN JE WILLIAMS MW SARVOTHAM SS
Citation: J. Vintenjohansen et al., REDUCTION IN INFARCT SIZE BUT NOT NEUTROPHIL ACCUMULATION BY CONTROLLED HYDRODYNAMICS OF REPERFUSION, The FASEB journal, 9(3), 1995, pp. 420-420
GRADUAL RESTORATION OF REPERFUSION REDUCES CORONARY-ARTERY ENDOTHELIAL INJURY AND MYOCARDIAL-INFARCTION
Authors: SATO H ZHAO ZO JORDAN JE WILLIAMS MW SARVOTHAM SS VINTENJOHANSEN J
Citation: H. Sato et al., GRADUAL RESTORATION OF REPERFUSION REDUCES CORONARY-ARTERY ENDOTHELIAL INJURY AND MYOCARDIAL-INFARCTION, The FASEB journal, 9(3), 1995, pp. 421-421
MONOCLONAL-ANTIBODY TO ICAM-1 REDUCES INFARCT SIZE FOLLOWING CORONARYOCCLUSION-REPERFUSION IN THE RABBIT
Authors: ZHAO ZQ LEFER DJ SATO H HART KK JEFFORDS PR BUDA AJ VINTENJOHANSEN J
Citation: Zq. Zhao et al., MONOCLONAL-ANTIBODY TO ICAM-1 REDUCES INFARCT SIZE FOLLOWING CORONARYOCCLUSION-REPERFUSION IN THE RABBIT, The FASEB journal, 9(3), 1995, pp. 421-421
POLYADENYLIC-ACID REDUCES INFARCT SIZE VIA INTRAVASCULAR ADENOSINE RECEPTOR MECHANISMS DURING REPERFUSION
Authors: TODD JC WILLIAMS MW ZHAO ZQ SATO H JORDAN JE VINTENJOHANSEN J
Citation: Jc. Todd et al., POLYADENYLIC-ACID REDUCES INFARCT SIZE VIA INTRAVASCULAR ADENOSINE RECEPTOR MECHANISMS DURING REPERFUSION, The FASEB journal, 9(3), 1995, pp. 421-421
AUGMENTATION OF MICROVASCULAR NITRIC-OXIDE IMPROVES MYOCARDIAL PERFORMANCE FOLLOWING GLOBAL-ISCHEMIA
Authors: HAMMON JW VINTENJOHANSEN J
Citation: Jw. Hammon et J. Vintenjohansen, AUGMENTATION OF MICROVASCULAR NITRIC-OXIDE IMPROVES MYOCARDIAL PERFORMANCE FOLLOWING GLOBAL-ISCHEMIA, Journal of cardiac surgery, 10(4), 1995, pp. 423-427
THE ROLE OF NITRIC-OXIDE AND NO-DONOR AGENTS IN MYOCARDIAL PROTECTIONFROM SURGICAL ISCHEMIC-REPERFUSION INJURY
Authors: VINTENJOHANSEN J SATO H ZHAO ZQ
Citation: J. Vintenjohansen et al., THE ROLE OF NITRIC-OXIDE AND NO-DONOR AGENTS IN MYOCARDIAL PROTECTIONFROM SURGICAL ISCHEMIC-REPERFUSION INJURY, International journal of cardiology, 50(3), 1995, pp. 273-281
DEPRESSION OF CARDIAC-FUNCTION AFTER DEEP HYPOTHERMIC CIRCULATORY ARREST IN DEEPLY ANESTHETIZED NEWBORN LAMBS
Authors: VELVIS H HINES MH KLOPFENSTEIN HS BERRY DD VINTENJOHANSEN J
Citation: H. Velvis et al., DEPRESSION OF CARDIAC-FUNCTION AFTER DEEP HYPOTHERMIC CIRCULATORY ARREST IN DEEPLY ANESTHETIZED NEWBORN LAMBS, Pediatric research, 37(4), 1995, pp. 36-36
SUPPLEMENTAL L-ARGININE DURING CARDIOPLEGIC ARREST AND REPERFUSION AVOIDS REGIONAL POSTISCHEMIC INJURY
Authors: SATO H ZHAO ZQ MCGEE DS WILLIAMS MW HAMMON JW VINTENJOHANSEN J
Citation: H. Sato et al., SUPPLEMENTAL L-ARGININE DURING CARDIOPLEGIC ARREST AND REPERFUSION AVOIDS REGIONAL POSTISCHEMIC INJURY, Journal of thoracic and cardiovascular surgery, 110(2), 1995, pp. 302-314
BLOOD CARDIOPLEGIA ENHANCED WITH NITRIC-OXIDE DONOR SPM-5185 COUNTERACTS POSTISCHEMIC ENDOTHELIAL AND VENTRICULAR DYSFUNCTION
Authors: NAKANISHI K ZHAO ZQ VINTENJOHANSEN J HUDSPETH DA MCGEE DS HAMMON JW
Citation: K. Nakanishi et al., BLOOD CARDIOPLEGIA ENHANCED WITH NITRIC-OXIDE DONOR SPM-5185 COUNTERACTS POSTISCHEMIC ENDOTHELIAL AND VENTRICULAR DYSFUNCTION, Journal of thoracic and cardiovascular surgery, 109(6), 1995, pp. 1146-1154
CONTROLLED HYDRODYNAMICS OF REPERFUSION REDUCES INFARCT SIZE INDEPENDENT OF DELAYED WASHOUT OF INTERSTITIAL ADENOSINE OR ATTENUATED NEUTROPHIL ACCUMULATION IN THE AREA AT RISK
Authors: VINTENJOHANSEN J SATO H ZHAO ZQ VANWYLEN DGL
Citation: J. Vintenjohansen et al., CONTROLLED HYDRODYNAMICS OF REPERFUSION REDUCES INFARCT SIZE INDEPENDENT OF DELAYED WASHOUT OF INTERSTITIAL ADENOSINE OR ATTENUATED NEUTROPHIL ACCUMULATION IN THE AREA AT RISK, Circulation, 92(8), 1995, pp. 2187-2187
Risultati: 1-25 | 26-50 | 51-75 | 76-77