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Results: 1-15 |
Results: 15
AXONAL INJURY ALTERS ALTERNATIVE SPLICING OF THE RETINAL NR1 RECEPTOR- THE PREFERENTIAL EXPRESSION OF THE NR1B ISOFORMS IS CRUCIAL FOR RETINAL GANGLION-CELL SURVIVAL
Authors: KREUTZ MR BOCKERS TM BOCKMANN J SEIDENBECHER CI KRACHT B VORWERK CK WEISE J SABEL BA
Citation: Mr. Kreutz et al., AXONAL INJURY ALTERS ALTERNATIVE SPLICING OF THE RETINAL NR1 RECEPTOR- THE PREFERENTIAL EXPRESSION OF THE NR1B ISOFORMS IS CRUCIAL FOR RETINAL GANGLION-CELL SURVIVAL, The Journal of neuroscience, 18(20), 1998, pp. 8278-8291
NITRATE THERAPY MAY RETARD GLAUCOMATOUS OPTIC NEUROPATHY, PERHAPS THROUGH MODULATION OF GLUTAMATE RECEPTORS
Authors: ZURAKOWSKI D VORWERK CK GORLA M KANELLOPOULOS AJ CHATURVEDI N GROSSKREUTZ CL LIPTON SA DREYER EB
Citation: D. Zurakowski et al., NITRATE THERAPY MAY RETARD GLAUCOMATOUS OPTIC NEUROPATHY, PERHAPS THROUGH MODULATION OF GLUTAMATE RECEPTORS, Vision research, 38(10), 1998, pp. 1489-1494
TIME-DEPENDENT ALTERATIONS OF VISUAL-EVOKED POTENTIALS AFTER OPTIC-NERVE CRUSH IN RATS
Authors: VORWERK CK HEIDUSCHKA P THANOS S SABEL BA KREUTZ MR
Citation: Ck. Vorwerk et al., TIME-DEPENDENT ALTERATIONS OF VISUAL-EVOKED POTENTIALS AFTER OPTIC-NERVE CRUSH IN RATS, Investigative ophthalmology & visual science, 38(4), 1997, pp. 4620-4620
ENDOTHELIAL NITRIC-OXIDE SYNTHASE IS NOT NECESSARY FOR THE FULL EXPRESSION OF EXCITOTOXICITY IN THE RETINA
Authors: GRAHAM KB VORWERK CK MILLER JW HUSAIN D DREYER EB
Citation: Kb. Graham et al., ENDOTHELIAL NITRIC-OXIDE SYNTHASE IS NOT NECESSARY FOR THE FULL EXPRESSION OF EXCITOTOXICITY IN THE RETINA, Investigative ophthalmology & visual science, 38(4), 1997, pp. 780-780
THE ROLE OF NEURONAL AND ENDOTHELIAL NITRIC-OXIDE SYNTHASE IN RETINALEXCITOTOXICITY
Authors: VORWERK CK HYMAN BT MILLER JW HUSAIN D ZURAKOWSKI D HUANG PL FISHMAN MC DREYER EB
Citation: Ck. Vorwerk et al., THE ROLE OF NEURONAL AND ENDOTHELIAL NITRIC-OXIDE SYNTHASE IN RETINALEXCITOTOXICITY, Investigative ophthalmology & visual science, 38(10), 1997, pp. 2038-2044
AXONAL INJURY ALTERS ALTERNATIVE SPLICING OF THE RETINAL NR1 RECEPTOR- THE PREFERENTIAL EXPRESSION OF THE NR1-B ISOFORMS IS CRUCIAL FOR RETINAL GANGLION-CELL SURVIVAL
Authors: KREUTZ MR SEIDENBECHER C BOCKERS TM BOCKMANN J VORWERK CK SABEL BA
Citation: Mr. Kreutz et al., AXONAL INJURY ALTERS ALTERNATIVE SPLICING OF THE RETINAL NR1 RECEPTOR- THE PREFERENTIAL EXPRESSION OF THE NR1-B ISOFORMS IS CRUCIAL FOR RETINAL GANGLION-CELL SURVIVAL, Journal of neurochemistry, 69, 1997, pp. 116-116
CHRONIC LOW-DOSE GLUTAMATE IS TOXIC TO RETINAL GANGLION-CELLS - TOXICITY BLOCKED BY MEMANTINE
Authors: VORWERK CK LIPTON SA ZURAKOWSKI D HYMAN BT SABEL BA DREYER EB
Citation: Ck. Vorwerk et al., CHRONIC LOW-DOSE GLUTAMATE IS TOXIC TO RETINAL GANGLION-CELLS - TOXICITY BLOCKED BY MEMANTINE, Investigative ophthalmology & visual science, 37(8), 1996, pp. 1618-1624
RETINAL GANGLION-CELL SUSCEPTIBILITY TO NMDA AND KAINATE EXCITOTOXICITY DEPENDS ON SOMA SIZE AND ECCENTRIC LOCATION
Authors: VORWERK CK BROSZ M BOCKERS TM SABEL BA KREUTZ MR
Citation: Ck. Vorwerk et al., RETINAL GANGLION-CELL SUSCEPTIBILITY TO NMDA AND KAINATE EXCITOTOXICITY DEPENDS ON SOMA SIZE AND ECCENTRIC LOCATION, Investigative ophthalmology & visual science, 37(3), 1996, pp. 695-695
RETINAL LOCALIZATION OF AN INOSITOL 1,3,4,5-TETRAKISPHOSPHATE RECEPTOR (INSP(4)R) AND EXPRESSION AFTER OPTIC-NERVE CRUSH
Authors: KREUTZ MR VORWERK CK BOCKERS TM HULSER E HUMPHREY M SABEL BA REISER G
Citation: Mr. Kreutz et al., RETINAL LOCALIZATION OF AN INOSITOL 1,3,4,5-TETRAKISPHOSPHATE RECEPTOR (INSP(4)R) AND EXPRESSION AFTER OPTIC-NERVE CRUSH, Investigative ophthalmology & visual science, 37(3), 1996, pp. 1512-1512
RETINAL GANGLION-CELLS FROM MICE DEFICIENT IN NEURONAL NITRIC-OXIDE SYNTHASE EXHIBIT NORMAL CALCIUM FLUXES IN RESPONSE TO GLUTAMATE
Authors: NGUYEN QD VORWERK CK DREYER EB
Citation: Qd. Nguyen et al., RETINAL GANGLION-CELLS FROM MICE DEFICIENT IN NEURONAL NITRIC-OXIDE SYNTHASE EXHIBIT NORMAL CALCIUM FLUXES IN RESPONSE TO GLUTAMATE, Investigative ophthalmology & visual science, 37(3), 1996, pp. 2900-2900
ETHAMBUTOL IS SELECTIVELY TOXIC TO RETINAL GANGLION-CELLS IN-VIVO
Authors: BHISITKUL RB VORWERK CK HENG JE VINALS AF DREYER EB
Citation: Rb. Bhisitkul et al., ETHAMBUTOL IS SELECTIVELY TOXIC TO RETINAL GANGLION-CELLS IN-VIVO, Investigative ophthalmology & visual science, 37(3), 1996, pp. 3575-3575
NEURONAL NITRIC-OXIDE SYNTHASE IS NECESSARY FOR THE FULL EXPRESSION OF EXCITOTOXICITY IN THE RETINA
Authors: YOO SH VORWERK CK MILLER JW HUSAIN D DREYER EB
Citation: Sh. Yoo et al., NEURONAL NITRIC-OXIDE SYNTHASE IS NECESSARY FOR THE FULL EXPRESSION OF EXCITOTOXICITY IN THE RETINA, Investigative ophthalmology & visual science, 37(3), 1996, pp. 3817-3817
SYSTEMIC L-KYNURENINE ADMINISTRATION PARTIALLY PROTECTS AGAINST NMDA,BUT NOT KAINATE-INDUCED DEGENERATION OF RETINAL GANGLION-CELLS, AND REDUCES VISUAL-DISCRIMINATION DEFICITS IN ADULT-RATS
Authors: VORWERK CK KREUTZ MR DREYER EB SABEL BA
Citation: Ck. Vorwerk et al., SYSTEMIC L-KYNURENINE ADMINISTRATION PARTIALLY PROTECTS AGAINST NMDA,BUT NOT KAINATE-INDUCED DEGENERATION OF RETINAL GANGLION-CELLS, AND REDUCES VISUAL-DISCRIMINATION DEFICITS IN ADULT-RATS, Investigative ophthalmology & visual science, 37(12), 1996, pp. 2382-2392
NMDA MEDIATED RETINAL GANGLION-CELL TOXICITY AS A FUNCTION OF CALCIUMAND MAGNESIUM CONCENTRATIONS
Authors: VORWERK CK MOSCARITOLA K DREYER EB
Citation: Ck. Vorwerk et al., NMDA MEDIATED RETINAL GANGLION-CELL TOXICITY AS A FUNCTION OF CALCIUMAND MAGNESIUM CONCENTRATIONS, Investigative ophthalmology & visual science, 36(4), 1995, pp. 935-935
EFFECTS OF BFGF AFTER RAT OPTIC-NERVE CRUSH
Authors: SCHMITT UW VORWERK CK SABEL BA
Citation: Uw. Schmitt et al., EFFECTS OF BFGF AFTER RAT OPTIC-NERVE CRUSH, Investigative ophthalmology & visual science, 35(4), 1994, pp. 1498-1498
Risultati: 1-15 |