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Zhang, YH
D'Souza, D
Raap, DK
Garcia, F
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Muma, NA
Van de Kar, LD
Citation: Yh. Zhang et al., Characterization of the functional heterologous desensitization of hypothalamic 5-HT1A receptors after 5-HT2A receptor activation, J NEUROSC, 21(20), 2001, pp. 7919-7927
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Van de Kar, LD
Javed, A
Zhang, YH
Serres, F
Raap, DK
Gray, TS
Citation: Ld. Van De Kar et al., 5-HT2A receptors stimulate ACTH, corticosterone, oxytocin, renin, and prolactin release and activate hypothalamic CRF and oxytocin-expressing cells, J NEUROSC, 21(10), 2001, pp. 3572-3579
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Battagli, G
Cabrera-Vera, TM
Van de Kar, LD
Citation: G. Battagli et al., Prenatal cocaine exposure potentiates 5-HT2A receptor function in male andfemale rat offspring, SYNAPSE, 35(3), 2000, pp. 163-172
Authors:
Newman, ME
Gur, E
Dremencov, E
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Citation: Me. Newman et al., Chronic clomipramine alters presynaptic 5-HT1B and postsynaptic 5-HT1A receptor sensitivity in rat hypothalamus and hippocampus, respectively, NEUROPHARM, 39(12), 2000, pp. 2309-2317
Authors:
Raap, DK
DonCarlos, L
Garcia, F
Muma, NA
Wolf, WA
Battaglia, G
Van de Kar, LD
Citation: Dk. Raap et al., Estrogen desensitizes 5-HT1A receptors and reduces levels of G(z), G(i1) and G(i3) proteins in the hypothalamus, NEUROPHARM, 39(10), 2000, pp. 1823-1832
Authors:
Serres, F
Muma, NA
Raap, DK
Garcia, F
Battaglia, G
Van de Kar, LD
Citation: F. Serres et al., Coadministration of 5-hydroxytryptamine(1A) antagonist WAY-100635 preventsfluoxetine-induced desensitization of postsynaptic 5-hydroxytryptamine(1A)receptors in hypothalamus, J PHARM EXP, 294(1), 2000, pp. 296-301
Authors:
Newman, ME
Li, Q
Gelfin, Y
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Lerer, B
Citation: Me. Newman et al., Low doses of ipsapirone increase growth hormone but not oxytocin secretionin normal male and female subjects, PSYCHOPHAR, 145(1), 1999, pp. 99-104
Authors:
Javed, A
Kamradt, MC
Van de Kar, LD
Gray, TS
Citation: A. Javed et al., D-fenfluramine induces serotonin-mediated Fos expression in corticotropin-releasing factor and oxytocin neurons of the hypothalamus, and serotonin-independent Fos expression in enkephalin and neurotensin neurons of the amygdala, NEUROSCIENC, 90(3), 1999, pp. 851-858
Authors:
Li, Q
Wichems, C
Heils, A
Van de Kar, LD
Lesch, KP
Murphy, DL
Citation: Q. Li et al., Reduction of 5-hydroxytryptamine (5-HT)(1A)-mediated temperature and neuroendocrine responses and 5-HT1A binding sites in 5-HT transporter knockout mice, J PHARM EXP, 291(3), 1999, pp. 999-1007
Authors:
Raap, DK
Garcia, F
Muma, NA
Wolf, WA
Battaglia, G
Van de Kar, LD
Citation: Dk. Raap et al., Sustained desensitization of hypothalamic 5-hydroxytryptamine(1A) receptors after discontinuation of fluoxetine: Inhibited neuroendocrine responses to 8-hydroxy-2-(dipropylamino)tetralin in the absence of changes in G(i/o/z)proteins, J PHARM EXP, 288(2), 1999, pp. 561-567
Authors:
Raap, DK
Evans, S
Garcia, F
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Muma, NA
Wolf, WA
Battaglia, G
Van de Kar, LD
Citation: Dk. Raap et al., Daily injections of fluoxetine induce dose-dependent desensitization of hypothalamic 5-HT1A receptors: Reductions in neuroendocrine responses to 8-OH-DPAT and in levels of G(z) and G(i) proteins, J PHARM EXP, 288(1), 1999, pp. 98-106