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Results: 1-15 |
Results: 15
PERTURBATION STABILITY OF THE ASYMMETRIC STOCHASTIC EQUILIBRIUM ASSIGNMENT MODEL
Authors: WATLING D
Citation: D. Watling, PERTURBATION STABILITY OF THE ASYMMETRIC STOCHASTIC EQUILIBRIUM ASSIGNMENT MODEL, Transportation research. Part B: methodological, 32(3), 1998, pp. 155-171
QUALITY-OF-LIFE IN MULTIPLE-SCLEROSIS IN FRANCE, GERMANY, AND THE UNITED-KINGDOM
Authors: MURPHY N CONFAVREUX C HAAS J KONIG N ROULLET E SAILER M SWASH M YOUNG C MEROT JL ZIMMERMANN C OWEN L WATLING D SHARIEF M BROHET S KANOUI V REGLING S BRAUN M TEWARI VK BOCHSLER JA AUGUIN C KAZEK MP COOK J TIFFONET F YEARDLEY H
Citation: N. Murphy et al., QUALITY-OF-LIFE IN MULTIPLE-SCLEROSIS IN FRANCE, GERMANY, AND THE UNITED-KINGDOM, Journal of Neurology, Neurosurgery and Psychiatry, 65(4), 1998, pp. 460-466
THE ROLE OF JAK STAT SIGNAL-TRANSDUCTION IN THE RESISTANCE OF MELANOMA TO INTERFERON TREATMENT/
Authors: JACKSON DP PATEL PM BANKS RE BURFOOT MS ROGERS NC WATLING D KERR IM SELBY PJ
Citation: Dp. Jackson et al., THE ROLE OF JAK STAT SIGNAL-TRANSDUCTION IN THE RESISTANCE OF MELANOMA TO INTERFERON TREATMENT/, British Journal of Cancer, 78, 1998, pp. 121-121
JAK2 AND STAT5, BUT NOT JAK1 AND STAT1, ARE REQUIRED FOR PROLACTIN-INDUCED BETA-LACTOGLOBULIN TRANSCRIPTION
Authors: HAN YL WATLING D ROGERS NC STARK GR
Citation: Yl. Han et al., JAK2 AND STAT5, BUT NOT JAK1 AND STAT1, ARE REQUIRED FOR PROLACTIN-INDUCED BETA-LACTOGLOBULIN TRANSCRIPTION, Molecular endocrinology, 11(8), 1997, pp. 1180-1188
A JAK1 JAK2 CHIMERA CAN SUSTAIN ALPHA-INTERFERON AND GAMMA-INTERFERONRESPONSES/
Authors: KOHLHUBER F ROGERS NC WATLING D FENG J GUSCHIN D BRISCOE J WITTHUHN BA KOTENKO SV PESTKA S STARK GR IHLE JN KERR IM
Citation: F. Kohlhuber et al., A JAK1 JAK2 CHIMERA CAN SUSTAIN ALPHA-INTERFERON AND GAMMA-INTERFERONRESPONSES/, Molecular and cellular biology, 17(2), 1997, pp. 695-706
JANUS KINASE-DEPENDENT ACTIVATION OF INSULIN-RECEPTOR-SUBSTRATE-1 IN RESPONSE TO INTERLEUKIN-4, ONCOSTATIN-M, AND THE INTERFERONS
Authors: BURFOOT MS ROGERS NC WATLING D SMITH JM PONS S PAONESSAW G PELLEGRINI S WHITE MF KERR IM
Citation: Ms. Burfoot et al., JANUS KINASE-DEPENDENT ACTIVATION OF INSULIN-RECEPTOR-SUBSTRATE-1 IN RESPONSE TO INTERLEUKIN-4, ONCOSTATIN-M, AND THE INTERFERONS, The Journal of biological chemistry, 272(39), 1997, pp. 24183-24190
JAK1 PLAYS AN ESSENTIAL ROLE FOR RECEPTOR PHOSPHORYLATION AND STAT ACTIVATION IN RESPONSE TO GRANULOCYTE-COLONY-STIMULATING FACTOR
Authors: SHIMODA K FENG J MURAKAMI H NAGATA S WATLING D ROGERS NC STARK GR KERR IM IHLE JN
Citation: K. Shimoda et al., JAK1 PLAYS AN ESSENTIAL ROLE FOR RECEPTOR PHOSPHORYLATION AND STAT ACTIVATION IN RESPONSE TO GRANULOCYTE-COLONY-STIMULATING FACTOR, Blood, 90(2), 1997, pp. 597-604
JAKS, STATS AND SIGNAL-TRANSDUCTION IN RESPONSE TO THE INTERFERONS AND OTHER CYTOKINES
Authors: BRISCOE J GUSCHIN D ROGERS NC WATLING D MULLER M HORN F HEINRICH P STARK GR KERR IM
Citation: J. Briscoe et al., JAKS, STATS AND SIGNAL-TRANSDUCTION IN RESPONSE TO THE INTERFERONS AND OTHER CYTOKINES, Philosophical transactions-Royal Society of London. Biological sciences, 351(1336), 1996, pp. 167-171
PLATELET-DERIVED GROWTH-FACTOR INDUCES PHOSPHORYLATION OF MULTIPLE JAK FAMILY KINASES AND STAT PROTEINS
Authors: VIGNAIS ML SADOWSKI HB WATLING D ROGERS NC GILMAN M
Citation: Ml. Vignais et al., PLATELET-DERIVED GROWTH-FACTOR INDUCES PHOSPHORYLATION OF MULTIPLE JAK FAMILY KINASES AND STAT PROTEINS, Molecular and cellular biology, 16(4), 1996, pp. 1759-1769
KINASE-NEGATIVE MUTANTS OF JAK1 CAN SUSTAIN INTERFERON-GAMMA-INDUCIBLE GENE-EXPRESSION BUT NOT AN ANTIVIRAL STATE
Authors: BRISCOE J ROGERS NC WITTHUHN BA WATLING D HARPUR AG WILKS A STARK GR IHLE JN KERR IM
Citation: J. Briscoe et al., KINASE-NEGATIVE MUTANTS OF JAK1 CAN SUSTAIN INTERFERON-GAMMA-INDUCIBLE GENE-EXPRESSION BUT NOT AN ANTIVIRAL STATE, EMBO journal, 15(4), 1996, pp. 799-809
ASYMMETRIC PROBLEMS AND STOCHASTIC-PROCESS MODELS OF TRAFFIC ASSIGNMENT
Authors: WATLING D
Citation: D. Watling, ASYMMETRIC PROBLEMS AND STOCHASTIC-PROCESS MODELS OF TRAFFIC ASSIGNMENT, Transportation research. Part B: methodological, 30(5), 1996, pp. 339-357
PARTICIPATION OF JAK AND STAT PROTEINS IN GROWTH HORMONE-INDUCED SIGNALING
Authors: HAN YL LEAMAN DW WATLING D ROGERS NC GRONER B KERR IM WOOD WI STARK GR
Citation: Yl. Han et al., PARTICIPATION OF JAK AND STAT PROTEINS IN GROWTH HORMONE-INDUCED SIGNALING, The Journal of biological chemistry, 271(10), 1996, pp. 5947-5952
A MAJOR ROLE FOR THE PROTEIN-TYROSINE KINASE JAK1 IN THE JAK STAT SIGNAL-TRANSDUCTION PATHWAY IN RESPONSE TO INTERLEUKIN-6/
Authors: GUSCHIN D ROGERS N BRISCOE J WITTHUHN B WATLING D HORN F PELLEGRINI S YASUKAWA K HEINRICH P STARK GR IHLE JN KERR IM
Citation: D. Guschin et al., A MAJOR ROLE FOR THE PROTEIN-TYROSINE KINASE JAK1 IN THE JAK STAT SIGNAL-TRANSDUCTION PATHWAY IN RESPONSE TO INTERLEUKIN-6/, EMBO journal, 14(7), 1995, pp. 1421-1429
URBAN TRAFFIC NETWORK MODELS AND DYNAMIC DRIVER INFORMATION-SYSTEMS
Authors: WATLING D
Citation: D. Watling, URBAN TRAFFIC NETWORK MODELS AND DYNAMIC DRIVER INFORMATION-SYSTEMS, Transport reviews, 14(3), 1994, pp. 219-246
COMPLEMENTATION BY THE PROTEIN-TYROSINE KINASE JAK2 OF A MUTANT-CELL LINE DEFECTIVE IN THE INTERFERON-GAMMA SIGNAL-TRANSDUCTION PATHWAY
Authors: WATLING D GUSCHIN D MULLER M SILVENNOINEN O WITTHUHN BA QUELLE FW ROGERS NC SCHINDLER C STARK GR IHLE JN KERR IM
Citation: D. Watling et al., COMPLEMENTATION BY THE PROTEIN-TYROSINE KINASE JAK2 OF A MUTANT-CELL LINE DEFECTIVE IN THE INTERFERON-GAMMA SIGNAL-TRANSDUCTION PATHWAY, Nature, 366(6451), 1993, pp. 166-170
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