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Results: 1-10 |
Results: 10
NF-KAPPA-B-MEDIATED INHIBITION OF APOPTOSIS IS REQUIRED FOR ENCEPHALOMYOCARDITIS VIRUS VIRULENCE - A MECHANISM OF RESISTANCE IN P50 KNOCKOUT MICE
Authors: SCHWARZ EM BADORFF C HIURA TS WESSELY R BADORFF A VERMA IM KNOWLTON KU
Citation: Em. Schwarz et al., NF-KAPPA-B-MEDIATED INHIBITION OF APOPTOSIS IS REQUIRED FOR ENCEPHALOMYOCARDITIS VIRUS VIRULENCE - A MECHANISM OF RESISTANCE IN P50 KNOCKOUT MICE, Journal of virology, 72(7), 1998, pp. 5654-5660
TRANSGENIC EXPRESSION OF REPLICATION-RESTRICTED ENTEROVIRAL GENOMES IN HEART-MUSCLE INDUCES DEFECTIVE EXCITATION-CONTRACTION COUPLING AND DILATED CARDIOMYOPATHY
Authors: WESSELY R KLINGEL K SANTANA LF DALTON N HONGO M LEDERER WJ KANDOLF R KNOWLTON KU
Citation: R. Wessely et al., TRANSGENIC EXPRESSION OF REPLICATION-RESTRICTED ENTEROVIRAL GENOMES IN HEART-MUSCLE INDUCES DEFECTIVE EXCITATION-CONTRACTION COUPLING AND DILATED CARDIOMYOPATHY, The Journal of clinical investigation, 102(7), 1998, pp. 1444-1453
LOW-LEVEL EXPRESSION OF A MUTANT COXSACKIEVIRAL CDNA INDUCES A MYOCYTOPATHIC EFFECT IN CULTURE - AN APPROACH TO THE STUDY OF ENTEROVIRAL PERSISTENCE IN CARDIAC MYOCYTES
Authors: WESSELY R HENKE A ZELL R KANDOLF R KNOWLTON KU
Citation: R. Wessely et al., LOW-LEVEL EXPRESSION OF A MUTANT COXSACKIEVIRAL CDNA INDUCES A MYOCYTOPATHIC EFFECT IN CULTURE - AN APPROACH TO THE STUDY OF ENTEROVIRAL PERSISTENCE IN CARDIAC MYOCYTES, Circulation, 98(5), 1998, pp. 450-457
COXSACKIEVIRUS B3 INDUCED CYTOPATHIC EFFECT IS INHIBITED IN-VITRO BY EXPRESSION OF A CLEAVAGE RESISTANT PROTEIN-SYNTHESIS INITIATION-FACTOR4G (EIF4G)
Authors: LEE GH WESSELY R KNOWLTON KU
Citation: Gh. Lee et al., COXSACKIEVIRUS B3 INDUCED CYTOPATHIC EFFECT IS INHIBITED IN-VITRO BY EXPRESSION OF A CLEAVAGE RESISTANT PROTEIN-SYNTHESIS INITIATION-FACTOR4G (EIF4G), Circulation, 98(17), 1998, pp. 3413-3413
TYPE-I BUT NOT TYPE-II INTERFERON RECEPTOR SIGNALING IS REQUIRED TO ATTENUATE COXSACKIEVIRAL VIRULENCE IN-VIVO
Authors: WESSELY R KLINGEL K KANDOLF R KNOWLTON KU
Citation: R. Wessely et al., TYPE-I BUT NOT TYPE-II INTERFERON RECEPTOR SIGNALING IS REQUIRED TO ATTENUATE COXSACKIEVIRAL VIRULENCE IN-VIVO, Circulation, 96(8), 1997, pp. 2362-2362
NF-KAPPA-B-MEDIATED INHIBITION OF APOPTOSIS IS ASSOCIATED WITH ENCEPHALOMYOCARDITIS VIRUS VIRULENCE IN-VIVO
Authors: BADORFF C SCHWARZ EM HIURA T WESSELY R KNOWLTON KU
Citation: C. Badorff et al., NF-KAPPA-B-MEDIATED INHIBITION OF APOPTOSIS IS ASSOCIATED WITH ENCEPHALOMYOCARDITIS VIRUS VIRULENCE IN-VIVO, Circulation, 96(8), 1997, pp. 2367-2367
COXSACKIEVIRAL PROTEASE 2A IS SUFFICIENT TO INDUCE A MYOCYTOPATHIC EFFECT THAT CAN HE RESCUED BY EXPRESSION OF A MUTANT ELF4G
Authors: WESSELY R BARRY L PALAKODETI R RHOADS RE KNOWLTON KU
Citation: R. Wessely et al., COXSACKIEVIRAL PROTEASE 2A IS SUFFICIENT TO INDUCE A MYOCYTOPATHIC EFFECT THAT CAN HE RESCUED BY EXPRESSION OF A MUTANT ELF4G, Circulation, 96(8), 1997, pp. 3190-3190
A MUTATION IN THE PUFF REGION OF VP2 ATTENUATES THE MYOCARDITIC PHENOTYPE OF AN INFECTIOUS CDNA OF THE WOODRUFF VARIANT OF COXSACKIEVIRUS B3
Authors: KNOWLTON KU JEON ES BERKLEY N WESSELY R HUBER S
Citation: Ku. Knowlton et al., A MUTATION IN THE PUFF REGION OF VP2 ATTENUATES THE MYOCARDITIC PHENOTYPE OF AN INFECTIOUS CDNA OF THE WOODRUFF VARIANT OF COXSACKIEVIRUS B3, Journal of virology, 70(11), 1996, pp. 7811-7818
LOW-LEVEL CARDIAC SPECIFIC EXPRESSION OF A NON INFECTIOUS COXSACKIEVIRAL CDNA LEADS TO CARDIOMYOPATHY IN TRANSGENIC MICE
Authors: WESSELY R KANDOLF R KNOWLTON KU
Citation: R. Wessely et al., LOW-LEVEL CARDIAC SPECIFIC EXPRESSION OF A NON INFECTIOUS COXSACKIEVIRAL CDNA LEADS TO CARDIOMYOPATHY IN TRANSGENIC MICE, Circulation, 94(8), 1996, pp. 3868-3868
EXPRESSION OF A MUTANT NONINFECTIOUS COXSACKIEVIRAL CDNA INHIBITS PROTEIN EXPRESSION IN CARDIAC MYOCYTES - A POTENTIAL MODEL OF VIRAL PERSISTENCE
Authors: WESSELY R HENKE A KANDOLF R KNOWLTON KU
Citation: R. Wessely et al., EXPRESSION OF A MUTANT NONINFECTIOUS COXSACKIEVIRAL CDNA INHIBITS PROTEIN EXPRESSION IN CARDIAC MYOCYTES - A POTENTIAL MODEL OF VIRAL PERSISTENCE, Circulation, 92(8), 1995, pp. 2515-2515
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