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KHASPEKOV L
SHAMLOO M
VICTOROV I
WIELOCH T
Citation: L. Khaspekov et al., SUBLETHAL IN-VITRO GLUCOSE-OXYGEN DEPRIVATION PROTECTS CULTURED HIPPOCAMPAL-NEURONS AGAINST A SUBSEQUENT SEVERE INSULT, NeuroReport, 9(7), 1998, pp. 1273-1276
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Authors:
CARDELL M
LANDSEND AS
EIDET J
WIELOCH T
BLACKSTAD TW
OTTERSEN OP
Citation: M. Cardell et al., HIGH-RESOLUTION IMMUNOGOLD ANALYSIS REVEALS DISTINCT SUBCELLULAR COMPARTMENTATION OF PROTEIN-KINASE-C-GAMMA AND PROTEIN-KINASE-DELTA IN RATPURKINJE-CELLS, Neuroscience, 82(3), 1998, pp. 709-725
Authors:
RUNDEN E
SEGLEN PO
HAUG FM
OTTERSEN OP
WIELOCH T
SHAMLOO M
LAAKE JH
Citation: E. Runden et al., REGIONAL SELECTIVE NEURONAL DEGENERATION AFTER PROTEIN PHOSPHATASE INHIBITION IN HIPPOCAMPAL SLICE CULTURES - EVIDENCE FOR A MAP KINASE-DEPENDENT MECHANISM, The Journal of neuroscience, 18(18), 1998, pp. 7296-7305
Authors:
FRIBERG H
FERRANDDRAKE M
BENGTSSON F
HALESTRAP AP
WIELOCH T
Citation: H. Friberg et al., CYCLOSPORINE-A, BUT NOT FK-506, PROTECTS MITOCHONDRIA AND NEURONS AGAINST HYPOGLYCEMIC DAMAGE AND IMPLICATES THE MITOCHONDRIAL PERMEABILITYTRANSITION IN CELL-DEATH, The Journal of neuroscience, 18(14), 1998, pp. 5151-5159
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Citation: F. Borismoller et T. Wieloch, CHANGES IN THE EXTRACELLULAR LEVELS OF GLUTAMATE AND ASPARTATE DURINGISCHEMIA AND HYPOGLYCEMIA - EFFECTS OF HYPOTHERMIA, Experimental Brain Research, 121(3), 1998, pp. 277-284
Citation: F. Kamme et T. Wieloch, INDUCTION OF JUND MESSENGER-RNA AFTER TRANSIENT FOREBRAIN ISCHEMIA INTHE RAT - EFFECT OF HYPOTHERMIA, Molecular brain research, 43(1-2), 1996, pp. 51-56
Authors:
SAVELAND H
NILSSON OG
BORISMOLLER F
WIELOCH T
BRANDT L
Citation: H. Saveland et al., INTRACEREBRAL MICRODIALYSIS OF GLUTAMATE AND ASPARTATE IN 2 VASCULAR TERRITORIES AFTER ANEURYSMAL SUBARACHNOID HEMORRHAGE, Neurosurgery, 38(1), 1996, pp. 12-19
Authors:
COIMBRA C
DRAKE M
BORISMOLLER F
WIELOCH T
Citation: C. Coimbra et al., LONG-LASTING NEUROPROTECTIVE EFFECT OF POSTISCHEMIC HYPOTHERMIA AND TREATMENT WITH AN ANTI-INFLAMMATORY ANTIPYRETIC DRUG - EVIDENCE FOR CHRONIC ENCEPHALOPATHIC PROCESSES FOLLOWING ISCHEMIA/, Stroke, 27(9), 1996, pp. 1578-1585
Authors:
DRAKE M
FRIBERG H
BORISMOLLER F
SAKATA K
WIELOCH T
Citation: M. Drake et al., THE IMMUNOSUPPRESSANT FK506 AMELIORATES ISCHEMIC DAMAGE IN THE RAT-BRAIN, Acta Physiologica Scandinavica, 158(2), 1996, pp. 155-159
Authors:
COIMBRA C
BORISMOLLER F
DRAKE M
WIELOCH T
Citation: C. Coimbra et al., DIMINISHED NEURONAL DAMAGE IN THE RAT-BRAIN BY LATE TREATMENT WITH THE ANTIPYRETIC DRUG DIPYRONE OR COOLING FOLLOWING CEREBRAL-ISCHEMIA, Acta Neuropathologica, 92(5), 1996, pp. 447-453
Citation: F. Kamme et al., BIPHASIC EXPRESSION OF THE FOS AND JUN FAMILIES OF TRANSCRIPTION FACTORS FOLLOWING TRANSIENT FOREBRAIN ISCHEMIA IN THE RAT - EFFECT OF HYPOTHERMIA, European journal of neuroscience, 7(10), 1995, pp. 2007-2016
Citation: Br. Hu et al., ALTERATIONS OF CA2+ CALMODULIN-DEPENDENT PROTEIN-KINASE-II AND ITS MESSENGER-RNA IN THE RAT HIPPOCAMPUS FOLLOWING NORMOTHERMIC AND HYPOTHERMIC ISCHEMIA/, Neuroscience, 68(4), 1995, pp. 1003-1016
Citation: Br. Hu et al., PERSISTENT TRANSLOCATION AND INHIBITION OF CA2+ CALMODULIN-DEPENDENT PROTEIN-KINASE-II IN THE CRUDE SYNAPTOSOMAL FRACTION OF THE VULNERABLEHIPPOCAMPUS FOLLOWING HYPOGLYCEMIA/, Journal of neurochemistry, 64(3), 1995, pp. 1361-1369
Citation: Br. Hu et T. Wieloch, PERSISTENT TRANSLOCATION OF CA2+ CALMODULIN-DEPENDENT PROTEIN-KINASE-II TO SYNAPTIC JUNCTIONS IN THE VULNERABLE HIPPOCAMPAL CA1 REGION FOLLOWING TRANSIENT ISCHEMIA/, Journal of neurochemistry, 64(1), 1995, pp. 277-284
Citation: J. Kurihara et al., CHANGES IN THE TYROSINE PHOSPHORYLATION OF MITOGEN-ACTIVATED PROTEIN-KINASE IN THE RAT HIPPOCAMPUS DURING AND FOLLOWING SEVERE HYPOGLYCEMIA, Journal of neurochemistry, 63(6), 1994, pp. 2346-2348
Citation: Br. Hu et T. Wieloch, TYROSINE PHOSPHORYLATION AND ACTIVATION OF MITOGEN-ACTIVATED PROTEIN-KINASE IN THE RAT-BRAIN FOLLOWING TRANSIENT CEREBRAL-ISCHEMIA, Journal of neurochemistry, 62(4), 1994, pp. 1357-1367
Citation: C. Coimbra et T. Wieloch, MODERATE HYPOTHERMIA MITIGATES NEURONAL DAMAGE IN THE RAT-BRAIN WHEN INITIATED SEVERAL HOURS FOLLOWING TRANSIENT CEREBRAL-ISCHEMIA, Acta Neuropathologica, 87(4), 1994, pp. 325-331
Citation: Br. Hu et al., HEAT-SHOCK INHIBITS PROTEIN-SYNTHESIS AND EIF-2 ACTIVITY IN CULTURED CORTICAL-NEURONS, Neurochemical research, 18(9), 1993, pp. 1003-1007
Citation: K. Bergstedt et T. Wieloch, CHANGES IN INSULIN-LIKE GROWTH FACTOR-I RECEPTOR DENSITY AFTER TRANSIENT CEREBRAL-ISCHEMIA IN THE RAT - LACK OF PROTECTION AGAINST ISCHEMICBRAIN-DAMAGE FOLLOWING INJECTION OF INSULIN-LIKE GROWTH FACTOR-I, Journal of cerebral blood flow and metabolism, 13(5), 1993, pp. 895-898
Citation: Br. Hu et T. Wieloch, STRESS-INDUCED INHIBITION OF PROTEIN-SYNTHESIS INITIATION - MODULATION OF INITIATION FACTOR-II AND GUANINE-NUCLEOTIDE EXCHANGE FACTOR ACTIVITIES FOLLOWING TRANSIENT CEREBRAL-ISCHEMIA IN THE RAT, The Journal of neuroscience, 13(5), 1993, pp. 1830-1838
Citation: Br. Hu et al., DEPRESSION OF NEURONAL PROTEIN-SYNTHESIS INITIATION BY PROTEIN-TYROSINE KINASE INHIBITORS, Journal of neurochemistry, 61(5), 1993, pp. 1789-1794
Citation: M. Cardell et T. Wieloch, TIME-COURSE OF THE TRANSLOCATION AND INHIBITION OF PROTEIN-KINASE-C DURING COMPLETE CEREBRAL-ISCHEMIA IN THE RAT, Journal of neurochemistry, 61(4), 1993, pp. 1308-1314
Citation: Br. Hu et T. Wieloch, CASEIN KINASE-II ACTIVITY IN THE POSTISCHEMIC RAT-BRAIN INCREASES IN BRAIN-REGIONS RESISTANT TO ISCHEMIA AND DECREASES IN VULNERABLE AREAS, Journal of neurochemistry, 60(5), 1993, pp. 1722-1728