Citation: As. Woolf et Pjd. Winyard, ADVANCES IN THE CELL BIOLOGY AND GENETICS OF HUMAN KIDNEY MALFORMATIONS, Journal of the American Society of Nephrology, 9(6), 1998, pp. 1114-1125
Authors:
ATTAR R
QUINN F
WINYARD PJD
MOURIQUAND PDE
FOXALL P
HANSON MA
WOOLF AS
Citation: R. Attar et al., SHORT-TERM URINARY FLOW IMPAIRMENT DEREGULATES PAX2 AND PCNA EXPRESSION AND CELL-SURVIVAL IN FETAL SHEEP KIDNEYS, The American journal of pathology, 152(5), 1998, pp. 1225-1235
Citation: Pjd. Winyard et al., EPITHELIAL GALECTIN-3 DURING HUMAN NEPHROGENESIS AND CHILDHOOD CYSTICDISEASES, Journal of the American Society of Nephrology, 8(11), 1997, pp. 1647-1657
Citation: Sa. Feather et al., ORAL-FACIAL-DIGITAL SYNDROME TYPE-1 IS ANOTHER DOMINANT POLYCYSTIC KIDNEY-DISEASE - CLINICAL, RADIOLOGICAL AND HISTOPATHOLOGICAL FEATURES OF A NEW KINDRED, Nephrology, dialysis, transplantation, 12(7), 1997, pp. 1354-1361
Citation: Sa. Feather et al., ORAL-FACIAL-DIGITAL SYNDROME TYPE-I IS ANOTHER DOMINANT POLYCYSTIC KIDNEY-DISEASE, Kidney international, 52(4), 1997, pp. 1131-1131
Authors:
KOLATSIJOANNOU M
MOORE R
WINYARD PJD
WOOLF AS
Citation: M. Kolatsijoannou et al., EXPRESSION OF HEPATOCYTE GROWTH-FACTOR SCATTER FACTOR AND ITS RECEPTOR, MET, SUGGESTS ROLES IN HUMAN EMBRYONIC ORGANOGENESIS, Pediatric research, 41(5), 1997, pp. 657-665
Citation: As. Woolf et al., GALECTIN-3 IN NORMAL POLYCYSTIC AND DYSPLASTIC NEPHROGENESIS, Journal of the American Society of Nephrology, 7(9), 1996, pp. 1886-1886
Authors:
WINYARD PJD
NAUTA J
LIRENMAN DS
HARDMAN P
SAMS VR
RISDON RA
WOOLF AS
Citation: Pjd. Winyard et al., DEREGULATION OF CELL-SURVIVAL IN CYSTIC AND DYSPLASTIC RENAL DEVELOPMENT, Kidney international, 49(1), 1996, pp. 135-146
Authors:
WINYARD PJD
RISDON RA
SAMS VR
DRESSLER GR
WOOLF AS
Citation: Pjd. Winyard et al., THE PAX2 TRANSCRIPTION FACTOR IS EXPRESSED IN CYSTIC AND HYPERPROLIFERATIVE DYSPLASTIC EPITHELIA IN HUMAN KIDNEY MALFORMATIONS, The Journal of clinical investigation, 98(2), 1996, pp. 451-459
Citation: Pjd. Winyard et al., WT1 EXPRESSION IN THE ABSENCE OF BCL2 MAY EXPLAIN EXCESSIVE APOPTOSISWHICH OCCURS IN HUMAN KIDNEY MALFORMATIONS, Journal of the American Society of Nephrology, 6(3), 1995, pp. 729-729
Authors:
DUKE VM
WINYARD PJD
THOROGOOD P
SOOTHILL P
BOULOUX PMG
WOOLF AS
Citation: Vm. Duke et al., KAL, A GENE MUTATED IN KALLMANNS-SYNDROME, IS EXPRESSED IN THE FIRST TRIMESTER OF HUMAN-DEVELOPMENT, Molecular and cellular endocrinology, 110(1-2), 1995, pp. 73-79
Authors:
WINYARD PJD
DUKE VM
THOROGOOD PV
NORMAN JT
BOULOUX PMG
WOOLF AS
Citation: Pjd. Winyard et al., RENAL APLASIA (RA) IN KALLMANNS-SYNDROME (KS) MAY BE EXPLAINED BY KALGENE-EXPRESSION DURING NEPHROGENESIS, Journal of the American Society of Nephrology, 5(3), 1994, pp. 640-640
Authors:
WINYARD PJD
LIRENMAN DS
RISDON RA
SAMS VR
WOOLF AS
Citation: Pjd. Winyard et al., WIDESPREAD APOPTOSIS OCCURS IN HUMAN RENAL MALFORMATIONS AND CHILDHOOD POLYCYSTIC KIDNEY-DISEASE (P K D), Journal of the American Society of Nephrology, 5(3), 1994, pp. 641-641