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Citation: Am. Samarel et al., Src and multiple MAP kinase activation in cardiac hypertrophy and congestive heart failure under chronic pressure-overload: Comparison with acute mechanical stretch, J MOL CEL C, 33(9), 2001, pp. 1637-1648
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Citation: Ra. Walsh et al., Process measures in an antenatal smoking cessation trial: another part of the picture, HEAL EDUC R, 15(4), 2000, pp. 469-483
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Citation: Ag. Schmidt et al., Cardiac-specific overexpression of calsequestrin results in left ventricular hypertrophy, depressed force-frequency relation and pulsus alternans in vivo, J MOL CEL C, 32(9), 2000, pp. 1735-1744
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Citation: Bd. Hoit et al., Influence of transgenic overexpression of phospholamban on postextrasystolic potentiation (vol 31, pg 2007, 1999), J MOL CEL C, 32(2), 2000, pp. 341-341
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Citation: Ag. Schmidt et al., Cardiac-specific overexpression of calsequestrin results in left ventricular hypertrophy, depressed force-frequency relation and pulsus alternans in vivo (vol 32, pg 1735, 2000), J MOL CEL C, 32(11), 2000, pp. 2105-2105
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Citation: Y. Ji et al., Disruption of a single copy of the SERCA2 gene results in altered Ca2+ homeostasis and cardiomyocyte function, J BIOL CHEM, 275(48), 2000, pp. 38073-38080
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Citation: Gu. Ahmmed et al., Changes in Ca2+ cycling proteins underlie cardiac action potential prolongation in a pressure-overloaded guinea pig model with cardiac hypertrophy and failure, CIRCUL RES, 86(5), 2000, pp. 558-570
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Citation: Y. Takeishi et al., Transgenic overexpression of constitutively active protein kinase C epsilon causes concentric cardiac hypertrophy, CIRCUL RES, 86(12), 2000, pp. 1218-1223