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Results: 1-6 |
Results: 6

Authors: Castilleja, A Ward, NE O'Brian, CA Swearingen, B Swan, E Gillogly, MA Murray, JL Kudelka, AP Gershenson, DM Ioannides, CG
Citation: A. Castilleja et al., Accelerated HER-2 degradation enhanced ovarian tumor recognition by CTL. Implications for tumor immunogenicity, MOL C BIOCH, 217(1-2), 2001, pp. 21-33

Authors: Chu, F Ward, NE O'Brian, CA
Citation: F. Chu et al., Potent inactivation of representative members of each PKC isozyme subfamily and PKD via S-thiolation by the tumor-promotion/progression antagonist glutathione but not by its precursor cysteine, CARCINOGENE, 22(8), 2001, pp. 1221-1229

Authors: Ward, NE Stewart, JR Ioannides, CG O'Brian, CA
Citation: Ne. Ward et al., Oxidant-induced S-gutathiolation inactivates protein kinase C-alpha (PKC-alpha): A potential mechanism of PKC isozyme regulation, BIOCHEM, 39(33), 2000, pp. 10319-10329

Authors: Ward, NE Fan, G O'Brian, CA
Citation: Ne. Ward et al., Differential non-redox inhibitory effects of glutathione against protein kinase C isozyme family members, ONCOL REP, 6(2), 1999, pp. 307-310

Authors: Stewart, JR Ward, NE Ioannides, CG O'Brian, CA
Citation: Jr. Stewart et al., Resveratrol preferentially inhibits protein kinase C-catalyzed phosphorylation of a cofactor-independent, arginine-rich protein substrate by a novel mechanism, BIOCHEM, 38(40), 1999, pp. 13244-13251

Authors: Ward, NE Pierce, DS Stewart, JR O'Brian, CA
Citation: Ne. Ward et al., A peptide substrate-based affinity label blocks protein kinase C-catalyzedATP hydrolysis and peptide-substrate phosphorylation, ARCH BIOCH, 365(2), 1999, pp. 248-253
Risultati: 1-6 |