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Results: 1-10 |
Results: 10
Deficiency or inhibition of Gas6 causes platelet dysfunction and protects mice against thrombosis
Authors: Angelillo-Scherrer, A de Frutos, PG Aparicio, C Melis, E Savi, P Lupu, F Arnout, J Dewerchin, M Hoylaerts, MF Herbert, M Collen, D Dahlback, B Carmeliet, P
Citation: A. Angelillo-scherrer et al., Deficiency or inhibition of Gas6 causes platelet dysfunction and protects mice against thrombosis, NAT MED, 7(2), 2001, pp. 215-221
The first laminin G-type domain in the SHBG-like region of protein S contains residues essential for activation of the receptor tyrosine kinase Sky
Authors: Evenas, P Dahlback, B de Frutos, PG
Citation: P. Evenas et al., The first laminin G-type domain in the SHBG-like region of protein S contains residues essential for activation of the receptor tyrosine kinase Sky, BIOL CHEM, 381(3), 2000, pp. 199-209
Protein S Thr103Asn mutation associated with type II deficiency reproducedin vitro and functionally characterised
Authors: Giri, TK de Frutos, PG Dahlback, B
Citation: Tk. Giri et al., Protein S Thr103Asn mutation associated with type II deficiency reproducedin vitro and functionally characterised, THROMB HAEM, 84(3), 2000, pp. 413-419
The second laminin G-type domain of protein S is indispensable for expression of full cofactor activity in activated protein C-catalysed inactivationof factor Va and factor VIIIa
Authors: Evenas, P de Frutos, PG Nicolaes, GAF Dahlback, B
Citation: P. Evenas et al., The second laminin G-type domain of protein S is indispensable for expression of full cofactor activity in activated protein C-catalysed inactivationof factor Va and factor VIIIa, THROMB HAEM, 84(2), 2000, pp. 271-277
Both G-type domains of protein S are required for the high-affinity interaction with C4b-binding protein (vol 266, pg 935, 1999)
Authors: Evenas, P de Frutos, PG Linse, S Dahlback, B
Citation: P. Evenas et al., Both G-type domains of protein S are required for the high-affinity interaction with C4b-binding protein (vol 266, pg 935, 1999), EUR J BIOCH, 267(2), 2000, pp. 625-625
Deficient APC-cofactor activity of protein S Heerlen in degradation of factor Va Leiden: a possible mechanism of synergism between thrombophilic riskfactors
Authors: Giri, TK Yamazaki, T Sala, N Dahlback, B de Frutos, PG
Citation: Tk. Giri et al., Deficient APC-cofactor activity of protein S Heerlen in degradation of factor Va Leiden: a possible mechanism of synergism between thrombophilic riskfactors, BLOOD, 96(2), 2000, pp. 523-531
Protein S secretion differences of missense mutants account for phenotypicheterogeneity
Authors: Espinosa-Parrilla, Y Yamazaki, T Sala, N Dahlback, B de Frutos, PG
Citation: Y. Espinosa-parrilla et al., Protein S secretion differences of missense mutants account for phenotypicheterogeneity, BLOOD, 95(1), 2000, pp. 173-179
Thrombophilia as a multigenic disease
Authors: Zoller, B de Frutos, PG Hillarp, A Dahlback, B
Citation: B. Zoller et al., Thrombophilia as a multigenic disease, HAEMATOLOG, 84(1), 1999, pp. 59-70
In vitro characterisation of two naturally occurring mutations in the thrombin-sensitive region of anticoagulant protein S
Authors: Girl, TK de Frutos, PG Yamazaki, T Villoutreix, BO Dahlback, B
Citation: Tk. Girl et al., In vitro characterisation of two naturally occurring mutations in the thrombin-sensitive region of anticoagulant protein S, THROMB HAEM, 82(6), 1999, pp. 1627-1633
Both G-type domains of protein S are required for the high-affinity interaction with C4b-binding protein
Authors: Evenas, P de Frutos, PG Linse, S Dahlback, B
Citation: P. Evenas et al., Both G-type domains of protein S are required for the high-affinity interaction with C4b-binding protein, EUR J BIOCH, 266(3), 1999, pp. 935-942
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