Authors: de Beer, F van Dijk, KW Jong, MC van Vark, LC van der Zee, A Hofker, MH Fallaux, FJ Hoeben, RC Smelt, AHM Havekes, LM

Citation: F. De Beer et al., Apolipoprotein E2 (Lys146 -> Gln) causes hypertriglyceridemia due to an apolipoprotein E variant-specific inhibition of lipolysis of very low densitylipoproteins-triglycerides, ART THROM V, 20(7), 2000, pp. 1800-1806

Authors: Jong, MC Hendriks, WL van Vark, LC Dahlmans, VEH Groener, JEM Havekes, LM

Citation: Mc. Jong et al., Oxidized VLDL induces less triglyceride accumulation in J774 macrophages than native VLDL due to an impaired extracellular lipolysis, ART THROM V, 20(1), 2000, pp. 144-151

Authors: Tacken, PJ de Beer, F van Vark, LC Havekes, LM Hofker, MH van Dijk, KW

Citation: Pj. Tacken et al., Very-low-density lipoprotein binding to the apolipoprotein E receptor 2 isenhanced by lipoprotein lipase, and does not require apolipoprotein E, BIOCHEM J, 347, 2000, pp. 357-361

Authors: Rensen, PCN Jong, MC van Vark, LC van der Boom, H Hendriks, WL van Berkel, TJC Biessen, EAL Havekes, LM

Citation: Pcn. Rensen et al., Apolipoprotein E is resistant to intracellular degradation in vitro and invivo - Evidence for retroendocytosis, J BIOL CHEM, 275(12), 2000, pp. 8564-8571

Authors: de Beer, F Hendriks, WL van Vark, LC Kamerling, SWA van Dijk, KW Hofker, MH Smelt, AHM Havekes, LM

Citation: F. De Beer et al., Binding of beta-VLDL to heparan sulfate proteoglycans requires lipoproteinlipase, whereas ApoE only modulates binding affinity, ART THROM V, 19(3), 1999, pp. 633-637