ACUTE MYOCARDIAL-INFARCTION DUE TO MUSCULAR BRIDGING OF THE LEFT ANTERIOR DESCENDING ARTERY - CLINICAL COURSE COMPLICATED BY CORONARY-ARTERY PERFORATION AFTER STENT IMPLANTATION - CASE-REPORT AND REVIEW OF THELITERATURE
D. Hering et al., ACUTE MYOCARDIAL-INFARCTION DUE TO MUSCULAR BRIDGING OF THE LEFT ANTERIOR DESCENDING ARTERY - CLINICAL COURSE COMPLICATED BY CORONARY-ARTERY PERFORATION AFTER STENT IMPLANTATION - CASE-REPORT AND REVIEW OF THELITERATURE, Zeitschrift fur Kardiologie, 86(8), 1997, pp. 630-638
A 47-year-old male patient was admitted to our hospital with acute ant
erior myocardial infarction. Immediate coronary angiography was carrie
d out, which showed proximal occlusion of the left anterior descending
artery (LAD). After mechanical recanalization, a reduction in vessel
caliber at the site of occlusion was visible, and balloon angioplasty
with consecutive stent implantation because of vessel wall dissection
was performed. After the procedure, diameter reduction of the entire v
essel segment distal to the stent and muscular bridging with subtotal
systolic obliteration of the LAD and one diagonal branch were demonstr
ated. Diastolic coronary flow did not appear to be limited (TIMI 3). D
ipyridamole-thallium cardiac imaging revealed an incomplete perfusion
defect of the anteroseptal region and a reversible perfusion reduction
of the anterolateral region. For definitive treatment, we decided to
implant a 3.0 mm-stent at the site of muscular bridging. Although ball
oon sizing was adapted to the diameter of the proximal reference segme
nt. measured by quantitative coronary angiography, coronary perforatio
n into the right ventricular outflow tract due to balloon oversizing i
n the distal dilation segment occurred. The patient remained asymptoma
tic at rest as well as under exercise testing, and hemodynamics remain
ed stable. Coronary re-angiography after 1 week demonstrated a persist
ent fistula with complete opacification of the LAD and normal coronary
flow (TIMI 3). Within the following 3 months, the coronary fistula cl
osed spontaneously. Conclusions: Muscular bridging is a ran cause of a
cute myocardial infarction. Balloon angioplasty and stent implantation
in the bridged segment may be complicated by coronary artery perforat
ion due to balloon oversizing. Risks and benefits of this therapeutic
option, therefore, have to be critically evaluated, and careful select
ion of balloon size using measurements of proximal and distal referenc
e diameter assessed hv intravascular ultrasound is recommended. Corona
ry artery perforation into the myocardium with subsequent development
of a fistula may be treated conservatively as long as the patient rema
ins asymptomatic. The frequency of spontaneous closure of the fistula
is high.