Various pathogenetic and functional morphologic aspects of renal trans
plant arteriopathy are presented and discussed with regard to Possible
parallels with ''conventional'' atherosclerosis, focusing on cellular
activities in the arterial wall and during local intramural lipid met
abolism. It becomes evident that, despite etiologic differences, the p
athogenetic course in renal transplant arteriopathy is manifested in a
n accelerated pattern largely similar to that of ''conventional'' athe
rosclerosis. These comparative studies provide important clues for the
understanding of similar arterial processes.