R. Lange et al., SURGICAL-TREATMENT OF TRICUSPID-VALVE END OCARDITIS - INDICATIONS, TECHNIQUE AND RESULTS, Zeitschrift fur Kardiologie, 84(11), 1995, pp. 921-929
Tricuspid valve endocarditis is treated by antibiotics alone in the ma
jority of the cases. However, intractable infection or hemodynamic com
promise may warrant surgery. In those cases total valve-excision or va
lve-replacement had been the most common surgical procedures. Both are
controversial in regards to the hemodynamic consequences and to the l
ong-term prognosis. In the following, results of tricuspid valve repai
r in acute infective endocarditis are reported and discussed as an add
itional treatment option. Between January 1988 and December 1993, 118
patients were operated for acute valve endocarditis at our institution
. Eleven of these patients had tricuspid valve endocarditis, isolated
(n = 7) or combined with endocarditis of a left-sided valve (n = 4). I
n the cases with isolated tricuspid valve endocarditis, the indication
for surgery was intractable infection in 6 and hemodynamically releva
nt tricuspid-insufficiency in 1 out of 7 patients, respectively. In al
l patients with associated left-sided endocarditis, the indication was
hemodynamic deterioration. In 8 patients the tricuspid valve endocard
itis was treated as follows: Debridement, vegectomy, patch-reconstruct
ion of the cusps, bicuspidalization. In 3 patients reconstruction was
not possible because of extended involvement of all parts of the valve
, including the valve ring and the papillary muscles. In these patient
s, primary valve-replacement (n = 1) or valve-excision with secondary
replacement (n = 2) was performed. In 4 patients tricuspid-reconstruct
ion was combined with mitral(n = 1), aortic- (n = 1) or double-valve r
eplacement (n = 2). 1 patient died early postoperatively in septic sho
ck and multi-organ failure after valve-excision. In the other patients
(n = 10) signs of infection vanished after surgery and tricuspid valv
e endocarditis healed without recurrences. In 7 patients late echocard
iographic follow-up showed tricuspid-regurgitation grade 0 in 3 patien
ts, I in 2, II in 1 and III in 1, respectively. If surgery is indicate
d, primary valve reconstruction is the treatment of choice for tricusp
id valve endocarditis. Only if extensive valve destruction excludes va
lve repair, would we now favor primary valve replacement over simple v
alvulectomy.