Rapid, clinical supraventricular tachycardia (SVT), without apparent u
nderlying heart disease, was identified in 3 young Labrador Retrievers
and 1 Labrador Retriever-type dog. The electrocardiographic character
istics, identification of ventricular preexcitation in 2 dogs, age at
onset, response to antiarrhythmic agents, and recent electrophysiologi
c documentation of a concealed accessory pathway in another young Labr
ador Retriever suggested orthodromic atrioventricular reciprocating ta
chycardia as the mechanism of SVT. As observed in this group of dogs,
chronic SVT can lead to systolic or diastolic dysfunction, overt conge
stive heart failure, or secondary valvular dysfunction, all of which a
re potentially reversible with control of the tachyarrhythmia. Intrave
nous administration of calcium-channel blockers appeared to be the mos
t effective antiarrhythmic treatment for acute termination of the tach
yarrhythmia in these dogs, whereas procainamide hydrochloride has been
most efficacious for chronic management. One dog died during direct-c
urrent cardioversion for incessant SVT; another was euthanatized becau
se of refractory congestive heart failure secondary to sustained SVT.