M. Terman et al., LIGHT TREATMENT FOR SLEEP DISORDERS - CONSENSUS REPORT .4. SLEEP PHASE AND DURATION DISTURBANCES, Journal of biological rhythms, 10(2), 1995, pp. 135-147
Advanced and delayed sleep phase disorders, and the hypersomnia that c
an accompany winter depression, have been treated successfully by appr
opriately timed artificial bright light exposure. Under entrainment to
the 24-h day-night cycle, the sleep-wake pattern may assume various p
hase relationships to the circadian pacemaker, as indexed, for example
, by abnormally long or short intervals between the onset of melatonin
production or the core body temperature minimum and wake-up time. Adv
anced and delayed sleep phase syndromes and non-24-h sleep-wake syndro
me have been variously ascribed to abnormal intrinsic circadian period
icity, deficiency of the entrainment mechanism, or-most simply-pattern
s of daily light exposure insufficient for adequate phase resetting. T
he timing of sleep is influenced by underlying circadian phase, but ps
ychosocial constraints also play a major role. Exposure to light early
or late in the subjective night has been used therapeutically to prod
uce corrective phase delays or advances, respectively, in both the sle
ep pattern and circadian rhythms. Supplemental light exposure in fall
and winter can reduce the hypersomnia of winter depression, although t
he therapeutic effect may be less dependent on timing.