Post-training cocaine intraperitoneal administration enhanced memory c
onsolidation in unoperated and sham-lesioned mice tested in a one-tria
l inhibitory avoidance task. Bilateral electrolytical amygdala lesions
blocked this effect. The results are interpreted in terms of involvem
ent of neurotransmitter, and in particular dopaminergic, systems in th
e effects of cocaine on memory.