Tinnitus is characterized by the continuous or intermittent auditory p
erception of various sounds (buzzing, whistling, etc.) in the absence
of any external stimulus. Perilymphatic hyperpressure is one of the nu
merous mechanisms which could hypothetically be involved in tinnitus g
eneration. In the present experiment, perilymphatic pressure was measu
red indirectly using the tympanic membrane displacement technique. Twe
nty-five tinnitus patients were investigated at 10, 15 and 20 dB above
the acoustic reflex threshold with ipsilateral stimulation. The varia
bles Vi (inward tympanic displacement), Vm (mean tympanic displacement
) and their variations according to stimulus level were compared betwe
en tinnitus sufferers and age-matched or hearing-matched controls. Tym
panic displacement was measured in sitting and supine positions so as
to evaluate cochlear aqueduct patency. No systematic changes in respon
se occurred in tinnitus patients, except at a high stimulation level,
perhaps due to hearing impairment.