The definition of the anatomical substrate of reentry in atrial flutte
r has allowed the recognition of narrow, critical areas of the circuit
, where radiofrequency ablation can interrupt reentry. In common fIutt
er the isthmus between the inferior vena cava and the tricuspid valve
appears the best target, but ablation between the coronary sinus and t
ricuspid valve can also be effective in some cases. In atypical flutte
r using the same circuit as common flutter in a ''clockwise'' directio
n, ablation of the same isthmus is effective. Flutter interruption is
the main objective, but it does not mean complete isthmus ablation. If
flutter remains inducible, new applications are delivered in the isth
mus, until it is made noninducible. Complications are rare. Despite at
taining noninducibility, flutter may recur, and new procedures may be
needed to prevent recurrence. Atrial fibrillation can occur in up to 3
0% of the cases during foIlow-up, but it is generally well controlled
with antiarrhythmic drugs, that were ineffective to treat flutter befo
re ablation. In reentry circuits based on surgical atrial scars, ablat
ion of an isthmus between the scar and the inferior vena cava can also
be effective. Left atrial circuits are not known well enough to guide
successful ablation.