R. Lange et al., TRICUSPID-VALVE RECONSTRUCTION, A TREATMENT OPTION IN ACUTE ENDOCARDITIS, European journal of cardio-thoracic surgery, 10(5), 1996, pp. 320-326
Tricuspid valve endocardititis is treated surgically by total valve ex
cision or valve replacement. Both procedures are controversial with re
gard to the hemodynamic consequences and to the long-term prognosis. I
n the following, results of tricuspid valve repair in acute infective
endocarditis are reported and discussed as an additional treatment opt
ion. Between January 1988 and December 1993, 118 patients were operate
d on for acute valve endocarditis at our institution. Eleven of these
patients had tricuspid valve endocarditis isolated (n = 7) or combined
with endocarditis of a left-sided valve (n = 4). In the cases with is
olated tricuspid valve endocarditis. the indication for surgery was in
tractable infection in six and hemodynamically relevant tricuspid insu
fficiency in one out of seven patients. In all patients with associate
d left-sided endocarditis, the indication was hemodynamic deterioratio
n. In eight patients the tricuspid valve endocarditis was treated as f
ollows: debridement, vegectomy, patch reconstruction of the cusps, red
ucing the cusps to two. In three patients reconstruction was not possi
ble because of extensive involvement of all parts of the valve, includ
ing the valve ring and the papillary muscles. In these patients primar
y valve replacement (n = 1) or valve excision with secondary replaceme
nt (n = 2) was performed. In four patients tricuspid reconstruction wa
s combined with mitral (n = 1), aortic (n = 1) or double valve replace
ment (n = 2). Postoperatively, signs of infection vanished in all surv
iving patients (n = 10) and tricuspid valve endocarditis healed withou
t recurrences. Implanted prosthetic material did not lead to recurrent
infection. One patient died early postoperatively after valve excisio
n, in septic shock and multi-organ failure. In seven patients late ech
ocardiographic follow-up showed tricuspid regurgitation grade 0 in thr
ee patients, I in two, II in one and III in one. Our results suggest t
hat valve repair is a reasonable treatment option for tricuspid valve
endocarditis in all cases with localized infection of the valve. Only
if extensive valve destruction excludes valve repair, would we now fav
or primary valve replacement over simple valvulectomy. In all other ca
ses primary valve reconstruction is the treatment of choice for tricus
pid valve endocarditis, if surgery is indicated.