Intracranial pressure often increases in animals with intracranial dis
ease. After intracranial compliance is exhausted, small increases in t
he volume of one of the components of intracranial tissue can cause dr
amatic increases in intracranial pressure. The clinical signs of incre
ased intracranial pressure are nonspecific. High intracranial pressure
can lead to decreases in cerebral perfusion or to lethal brain hernia
tion. The threshold at which intracranial pressure becomes pathologic
has not been established. Intracranial pressure in a dog or cat can be
monitored with a fiberoptic system. Various methods have been used to
reduce intracranial pressure, including head elevation, diuretics, hy
perventilation, aspiration of cerebrospinal fluid, barbiturates, hypot
hermia, and craniotomy. Whether corticosteroids are helpful is controv
ersial. No clear consensus exists on how and when to treat animals tha
t are suspected to have increased intracranial pressure. Recognizing t
he subtle clinical changes that occur with increasing intracranial pre
ssure and treating increased intracranial pressure early in the course
of the disease offer the best chance of recovery.