We report a patient with prerectal eyelid lag without retraction. MRI
showed high signal abnormality dorsal to the right red nucleus in a re
gion thought responsible for premotor eyelid control. The dissociation
of eyelid lag from eyelid retraction in this patient would suggest th
at these lid signs may have separate neural mechanisms or pathways. We
propose that the lesion interrupts inhibitory connections between the
vertical gaze center and the central caudal nucleus.