A 26-year-old man acutely developed bilateral sensory symptoms of carp
al tunnel syndrome (CTS). Neurophysiological investigations confirmed
the diagnosis of CTS with findings suggesting conduction block of sens
ory fibers. Endocrinological studies revealed a hyperthyroidism. He wa
s treated with methimazole and propranolol with improvement of thyroid
function. The neurophysiological and clinical follow-up revealed a pr
ogressive improvement of CTS. The echography performed at mist bilater
ally failed to show any rough compressive factors in the carpel tunnel
. The clinical, neurophysiological and endocrinological evolution sugg
est a relationship between hyperthyroidism and acute bilateral CTS.