Thirty-seven temporal bones were dissected and the posterior tympanic
and epitympanic folds recorded and photographed. Histologic details we
re documented from four serially sectioned temporal bones, two normal
and two inflamed. Of these 41 specimens, 31 were normal, and 10 showed
signs of inflammation. The type for the lateral fold was incudomallea
l in 16 and incudal in 25 ears. Incus intercrural and incudostapedial
folds appeared only exceptionally. Medial and superior incudal folds w
ere not present in normal ears. The anterior tympanic isthmus was a co
nstant, large aeration pathway. In chronically inflamed ears, its part
ial or total block was caused by polypoid or large sheet-like folds. I
nactive sequelae appeared as mature, simple, one-layer or extensive mu
ltilayer networks of webs, connected with a deeply indrawn incudomalle
al fold. The small posterior isthmus was open to the incudal fossa in
13 ears, and in 28, it was sealed off by a posterior incudal fold. The
mastoid air cell tracks were (in ten of 37 dissected ears) open to th
e incudal fossa, or directly, to the posterior tympanum. Auxiliary pat
hways due to membrane defects were found in both the horizontal and de
scending portions of the incudomalleal fold. Excepting the chordal, in
cudomalleal, and posterior incudal folds, fold-like webs in the poster
ior tympanum and epitympanum are of inflammatory origin.