There is controversy over the suggestion that recanalisation of extrac
ranially occluded internal carotid arteries (ICAs) is more frequent th
an actually documented, A follow-up of thirty patients with sonographi
cally (n = 31) and angiographically (n = 29) verified occlusion of the
ICA was performed at median 401 days after occlusion. In 24 cases, th
e occlusion was due to arteriosclerosis and thrombosis; in three cases
due to macroembolism; in three cases due to dissection and in one cas
e due to exogenous trauma. 16 patients received an anticoagulant thera
py with intravenous heparin (25 000 IE/d). In three patients with arte
riosclerotic-thrombotic or embolic occlusion of the ICA partial or com
plete recanalisation was found 18 days, 37 days and 16 years respectiv
ely after the initial diagnosis. Two of them received high-dose intrav
enous heparin. Two patients with occlusions due to dissection showed n
early normalized recanalisation. They were treated with high-dose hepa
rin, Our results show that recanalisation of an occluded carotid arter
y due to arteriosclerosis and thrombosis or macroembolism is possible
but rare. Treatment with high-dose heparin does not lead to better rec
analisation. Occlusion due to dissection shows a higher rate of recana
lisation.