CEREBELLAR HEMORRHAGIC INFARCTION

We investigated 17 patients with 26 cerebellar hemorrhagic infarcts fo r their vascular anatomy, stroke mechanisms, and clinical course. Sixt een infarcts involved the superior cerebellar artery, nine the posteri or inferior cerebellar artery, and one the anterior inferior cerebella r artery territories. The infarcts involved the full territory of the supplying arteries in 19 of 26 infarcts (73%). Sixteen of 17 patients were stable or improving when the hemorrhagic infarction was detected. All but one patient had an imaging study at the time of presentation that was negative for blood; hemorrhagic infarction was detected on ro utine serial scans performed during the first 15 days. Nine of the 17 patients were on anticoagulants when the cerebellar hemorrhagic infarc t was detected; anticoagulation was maintained in eight of them with n o clinical worsening. The stroke mechanism in all patients was conside red embolic from cardiac and intraarterial sources. The causes, imagin g findings, and consequences of hemorrhagic infarcts in the posterior circulation are similar to those in the anterior circulation.