Clinical signs of tracheal collapse usually are seen in young calves e
xhibiting inspiratory distress. The inspiratory dyspnea causes strider
in the calves' breathing and a marked increase in the duration of the
inspiratory phase of respiration. Calves breathe with their mouth ope
n and head and neck extended, but body temperatures are seldom elevate
d more than 1 degrees to 1.5 degrees C above normal. Auscultation of t
he respiratory tract shows harsh, often bubbly, tracheal sounds that a
re referred into the chest. Calves can be stressed easily to the point
of collapse by even the simplest restraint procedures, so extreme car
e must be taken during examination. Historically, most calves with tra
cheal collapse have been cases of dystocia at birth. Although evidence
of the collapse seldom is seen from birth, it is thought that the ini
tiating injury to the trachea occurs at that time. Compression of the
anterior chest wall with fracturing of the first few pairs of ribs may
cause injury to the tracheal rings at the thoracic inlet. Because of
the elastic nature of the rings, they tend to spring back to a circula
r shape, but the compressed rings are left less rigid and have the ten
dency to malformation in response to pressure changes in the chest and
trachea. As the calf ages, either of two events may contribute to a g
radual worsening of the condition-exuberant callus formation around th
e rib fractures compresses the trachea at the thoracic inlet or the th
oracic segment of the trachea weakens secondarily to the increased neg
ative pressure exerted by the calf during inspiration (dynamic collaps
e).