CAN OBSTRUCTIVE SLEEP-APNEA BE A COMPLICATION OF UVULOPALATOPHARYNGOPLASTY

Citation
Y. Finkelstein et al., CAN OBSTRUCTIVE SLEEP-APNEA BE A COMPLICATION OF UVULOPALATOPHARYNGOPLASTY, Journal of Laryngology and Otology, 109(3), 1995, pp. 212-217
Citations number
30
Categorie Soggetti
Otorhinolaryngology
ISSN journal
00222151
Volume
109
Issue
3
Year of publication
1995
Pages
212 - 217
Database
ISI
SICI code
0022-2151(1995)109:3<212:COSBAC>2.0.ZU;2-D
Abstract
Polysomnography is currently used for diagnosis, evaluation and select ion of therapy in patients with obstructive sleep apnoea (OSA), but cl inically successful uvulopalatopharyngoplasty (UPPP) is not necessaril y reflected by post-operative improvement of polysomnographic recordin gs. Post-operative polysomnography may suggest deterioration of pre-ex isting OSA or, in snorers, de-novo precipitation of OSA. Thus, if poly somnography is a reliable indicator of OSA, then OSA may be a post-ope rative risk of UPPP. The aims of our study were: (i) to assess the pos sible deleterious effect of UPPP on sleep patterns; (ii) to further de fine the role of cardioisotope scanning in the evaluation of OSA; (iii ) to assess the reliability of polysomnography given the clinical and cardioisotope scan findings. Symptoms, polysomnography and radionuclid e ventriculography were prospectively compared pre- and post-operative ly in 41 patients undergoing UPPP. In 12 patients (29 per cent), there were disparate results between pre- or post-operative polysomnography and the clinical and/or radionuclide ventriculography, as follows: In four of 16 patients with abnormal pre-operative ventricular performan ce, there was pre-operative symptomatology of severe OSA and a bedmate 's reports of apnoeic episodes. This was in contrast to normal or near normal sleep apnoea recordings. In eight patients, post-operative imp rovement of symptoms was reported, despite deterioration of post-opera tive polysomnographic recordings. In these patients the post-operative improvement of symptoms was also reflected by improved ventricular pe rformance. Worsening of ventricular performance was not demonstrated i n any patient. In conclusion, UPPP does not induce OSA. Polysomnograph y may underestimate or even misdiagnose cases of OSA. The diagnostic i mportance of patient symptomatology should be stressed particularly in those patients with only mildly abnormal or even completely normal sl eep studies. A combination of polysomnographic and cardiovascular eval uation in patients with symptomatology consistent with OSA is recommen ded.