In 1993, a wild mustard population growing in a canola crop near Wetas
kiwin, AB was poorly controlled by ethametsulfuron after only one prev
ious use of the herbicide. Controlled environment experiments were con
ducted to compare the response of this suspected resistant population
(R) with that of a known susceptible population (S) collected near Veg
reville, AB to increasing rates of ethametsulfuron, metsulfuron, chlor
sulfuron, thifensulfuron, HOE 075032, imazamethabenz, imazethapyr, met
ribuzin, and 2,4-D. The R wild mustard population was highly resistant
to ethametsulfuron, slightly resistant to low rates of metsulfuron bu
t not resistant to any of the other herbicides tested. This suggests t
hat the mechanism of resistance may differ from that reported for othe
r sulfonylurea and imidazolinone herbicides. Since resistance was docu
mented after only 2 yr of ethametsulfuron use, the initial frequency o
f resistance to this herbicide in wild mustard populations may be very
high. Dry weight of untreated plants did not differ significantly bet
ween the S and R populations suggesting little or no differences in co
mpetitiveness between them.